作为牙周病发病机制中的一个因素，补体通过替代途径激活。
Activation of complement by the alternative pathway as a factor in the pathogenesis of periodontal disease.
作者信息
Allison A C, Schorlemmer H U
出版信息
Lancet. 1976 Nov 6;2(7993):1001-4. doi: 10.1016/s0140-6736(76)90837-0.
PMID:62218
Abstract
Dental plaque and a bacterium, Actinomyces viscosus, isolated from plaque that can reproduce periodontal disease in germ-free rats, are activators of complement by the alternative pathway. It is suggested that this process is involved in the pathogenesis of chronic inflammatory periodontal disease.
摘要
牙菌斑以及从牙菌斑中分离出的一种细菌——黏性放线菌，该细菌可在无菌大鼠身上引发牙周病，它们是补体替代途径的激活剂。有人提出，这一过程与慢性炎症性牙周病的发病机制有关。
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