水貂细胞病灶诱导型鼠C型病毒基因组分析：进展报告 - Suppr

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超能文献

Analysis of the genomes of mink cell focus-inducing murine type-C viruses: a progress report.

作者信息

Lung M L, Hering C, Hartley J W, Rowe W P, Hopkins N

出版信息

Cold Spring Harb Symp Quant Biol. 1980;44 Pt 2,:1269-74. doi: 10.1101/sqb.1980.044.01.138.

DOI:10.1101/sqb.1980.044.01.138

PMID:6253201

Abstract

摘要

相似文献

Analysis of the genomes of mink cell focus-inducing murine type-C viruses: a progress report.水貂细胞病灶诱导型鼠C型病毒基因组分析：进展报告

Cold Spring Harb Symp Quant Biol. 1980;44 Pt 2,:1269-74. doi: 10.1101/sqb.1980.044.01.138.

Large RNase T1-resistant oligonucleotides encoding p15E and the U3 region of the long terminal repeat distinguish two biological classes of mink cell focus-forming type C viruses of inbred mice.编码p15E和长末端重复序列U3区域的大型核糖核酸酶T1抗性寡核苷酸区分了近交系小鼠中貂细胞集落形成C型病毒的两种生物学类别。

J Virol. 1983 Jan;45(1):275-90. doi: 10.1128/JVI.45.1.275-290.1983.

Origin of mink cytopathic focus-forming (MCF) viruses:comparison with ecotropic and xenotropic murine leukemia virus genomes.水貂细胞病变灶形成（MCF）病毒的起源：与嗜亲性和异嗜性鼠白血病病毒基因组的比较。

Virology. 1981 Sep;113(2):465-83. doi: 10.1016/0042-6822(81)90175-6.

Status of the association of mink cell focus-forming viruses with leukemogenesis.水貂细胞致细胞病变病毒与白血病发生的关联状况。

Cold Spring Harb Symp Quant Biol. 1980;44 Pt 2,:1265-8. doi: 10.1101/sqb.1980.044.01.137.

Isolation and characterization of mink cell focus-inducing murine leukemia viruses with xenotropic host range from mouse strain SL.从小鼠品系SL中分离并鉴定具有嗜异性宿主范围的水貂细胞灶诱导型鼠白血病病毒。

J Virol. 1983 Jan;45(1):447-51. doi: 10.1128/JVI.45.1.447-451.1983.

Cellular origin and role of mink cell focus-forming viruses in murine thymic lymphomas.水貂细胞集落形成病毒在小鼠胸腺淋巴瘤中的细胞起源及作用

Nature. 1982 Jan 7;295(5844):25-31. doi: 10.1038/295025a0.

A Mus dunni cell line that lacks sequences closely related to endogenous murine leukemia viruses and can be infected by ectropic, amphotropic, xenotropic, and mink cell focus-forming viruses.一种缺乏与内源性鼠白血病病毒密切相关序列的邓氏小鼠细胞系，可被嗜亲性、双嗜性、异嗜性和貂细胞集落形成病毒感染。

J Virol. 1984 Nov;52(2):695-8. doi: 10.1128/JVI.52.2.695-698.1984.

Restriction of murine leukemia viruses by Fv-1: a model for studying host genetic control of retroviral gene movement and leukemogenesis.Fv-1对鼠白血病病毒的限制：研究逆转录病毒基因转移和白血病发生的宿主遗传控制的模型

Prog Nucleic Acid Res Mol Biol. 1983;29:175-92. doi: 10.1016/s0079-6603(08)60446-8.

Lymphomagenicity of recombinant mink cell focus-inducing murine leukemia viruses.重组貂细胞集落形成诱导型鼠白血病病毒的淋巴瘤致瘤性

J Exp Med. 1980 Mar 1;151(3):542-52. doi: 10.1084/jem.151.3.542.

Comparative analysis of RNA tumor virus genomes.RNA肿瘤病毒基因组的比较分析

Haematol Blood Transfus. 1979;23:529-52. doi: 10.1007/978-3-642-67057-2_70.

引用本文的文献

Duplication of U3 sequences in the long terminal repeat of mink cell focus-inducing viruses generates redundancies of transcription factor binding sites important for the induction of thymomas.水貂细胞融合诱导病毒长末端重复序列中U3序列的重复产生了对胸腺瘤诱导至关重要的转录因子结合位点冗余。

J Virol. 2003 Mar;77(5):3326-33. doi: 10.1128/jvi.77.5.3326-3333.2003.

A direct demonstration of recombination between an injected virus and endogenous viral sequences, resulting in the generation of mink cell focus-inducing viruses in AKR mice.注射病毒与内源性病毒序列之间发生重组的直接证据，这导致在AKR小鼠中产生了貂细胞融合诱导病毒。

J Virol. 1993 Jul;67(7):3763-70. doi: 10.1128/JVI.67.7.3763-3770.1993.

Genetic study of lymphoma induction by AKR mink cell focus-inducing virus in AKR x NFS crosses.AKR水貂细胞致瘤病毒在AKR×NFS杂交中诱导淋巴瘤的遗传学研究。

J Exp Med. 1981 Aug 1;154(2):450-7. doi: 10.1084/jem.154.2.450.

Analysis of the env gene of a molecularly cloned and biologically active Moloney mink cell focus-forming proviral DNA.对分子克隆且具有生物活性的莫洛尼貂细胞病灶形成前病毒DNA的env基因进行分析。

J Virol. 1982 Oct;44(1):19-31. doi: 10.1128/JVI.44.1.19-31.1982.

Structure of retroviral RNAs produced by cell lines derived from spontaneous lymphomas of AKR mice.源自AKR小鼠自发性淋巴瘤的细胞系所产生的逆转录病毒RNA的结构

J Virol. 1982 Jan;41(1):18-29. doi: 10.1128/JVI.41.1.18-29.1982.

New cell surface gp70 related to Friend mink cell focus-inducing virus is expressed on Friend virus-induced erythroleukemic spleen cells after elimination of ecotropic Friend virus gp70 in Rfv-3r/s mice.与弗氏水貂细胞灶诱导病毒相关的新细胞表面糖蛋白70，在Rfv-3r/s小鼠中消除嗜异性弗氏病毒糖蛋白70后，表达于弗氏病毒诱导的红白血病脾细胞上。

J Exp Med. 1981 Sep 1;154(3):868-82. doi: 10.1084/jem.154.3.868.

Protection from spontaneous lymphoma development in SJL/J(v+) mice neonatally injected with dualtropic SJL-151 virus.新生期注射双嗜性SJL-151病毒的SJL/J(v+)小鼠对自发性淋巴瘤发展的保护作用。

Proc Natl Acad Sci U S A. 1983 May;80(9):2775-9. doi: 10.1073/pnas.80.9.2775.

Generation of AKR mink cell focus-forming viruses: a conserved single-copy xenotrope-like provirus provides recombinant long terminal repeat sequences.AKR水貂细胞灶形成病毒的产生：一个保守的单拷贝异种嗜性样前病毒提供重组长末端重复序列。

J Virol. 1984 May;50(2):432-8. doi: 10.1128/JVI.50.2.432-438.1984.

Envelope gene sequence of two in vitro-generated mink cell focus-forming murine leukemia viruses which contain the entire gp70 sequence of the endogenous nonecotropic parent.两种体外产生的貂细胞灶形成性鼠白血病病毒的包膜基因序列，这些病毒包含内源性非嗜异性亲本的完整gp70序列。

J Virol. 1984 Feb;49(2):530-9. doi: 10.1128/JVI.49.2.530-539.1984.

Unusual properties of AKR MCF-247 murine leukemia virus unintegrated proviral DNA.AKR MCF-247鼠白血病病毒未整合前病毒DNA的异常特性。

Arch Virol. 1983;78(3-4):255-66. doi: 10.1007/BF01311320.

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