病毒介导的通透性变化研究
Survey of virally mediated permeability changes.
作者信息
Foster K A, Gill K, Micklem K J, Pasternak C A
出版信息
Biochem J. 1980 Sep 15;190(3):639-46. doi: 10.1042/bj1900639.
Abstract
- Sendai virus causes permeability changes when added to freshly isolated brain cells (cerebellum or ependymal cells) or to a culture of forebrain cells. 2. Sendai virus causes permeability changes when added to organ cultures of ferret lung or nasal turbinate. Influenza virus causes no permeability changes under these conditions. 3. Rabies virus and vesicular-stomatitis virus, in contrast with Sendai virus, do not cause permeability changes in BHK cells or Lettrée cells. 4. Serum from patients suffering from viral hepatitis does not cause permeability changes in human leucocytes; addition to Sendai virus causes permeability changes. 5. It is concluded that permeability changes accompanying viral entry occur only with certain types of paramyxovirus, but that there is little restriction on cell type. 6. MDBK cells infected with Sendai virus show permeability changes during viral release, similar to those that occur during viral entry. Because these changes do not appear to be restricted to paramyxoviruses, they may have considerable clinical significance.
摘要
- 将仙台病毒添加到新分离的脑细胞（小脑或室管膜细胞）或前脑细胞培养物中时，会引起通透性变化。2. 将仙台病毒添加到雪貂肺或鼻甲的器官培养物中时，会引起通透性变化。在这些条件下，流感病毒不会引起通透性变化。3. 与仙台病毒相反，狂犬病病毒和水疱性口炎病毒不会在BHK细胞或莱特里细胞中引起通透性变化。4. 病毒性肝炎患者的血清不会在人白细胞中引起通透性变化；添加到仙台病毒中会引起通透性变化。5. 得出的结论是，伴随病毒进入的通透性变化仅在某些类型的副粘病毒中出现，但对细胞类型几乎没有限制。6. 感染仙台病毒的MDBK细胞在病毒释放期间显示出通透性变化，类似于病毒进入期间发生的变化。由于这些变化似乎并不局限于副粘病毒，它们可能具有相当大的临床意义。
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