多个基因对细菌多糖抗原抗体反应强度的影响。
Influence of multiple genes on the magnitude of the antibody response to bacterial polysaccharide antigens.
作者信息
Baker P J, Rudbach J A, Prescott B, Caldes G, Evans C, Stashak P W
出版信息
Infect Immun. 1984 Jul;45(1):56-61. doi: 10.1128/iai.45.1.56-61.1984.
Abstract
Studies conducted with F1 and F2 progeny of crosses between strains of inbred mice that differ greatly in their capacity to make an antibody response to type III pneumococcal polysaccharide, dextran B-1355, and lipopolysaccharide from Escherichia coli 0113 have shown that multiple genes influence the magnitude of the antibody response to these antigens. Other studies with hybrids derived from crosses between C3H/HeJ, CBA/N, and RIIIS/J mice have indicated that the genetic defects characteristic of these strains of mice are dissimilar and unlinked and that autosomal, as well as X-linked, genes control serum immunoglobulin M in unimmunized mice.
摘要
对在产生针对III型肺炎球菌多糖、葡聚糖B - 1355和大肠杆菌0113脂多糖的抗体反应能力上差异很大的近交系小鼠品系间杂交的F1和F2后代进行的研究表明，多个基因影响对这些抗原的抗体反应强度。对来自C3H/HeJ、CBA/N和RIIIS/J小鼠杂交产生的杂种进行的其他研究表明，这些小鼠品系的遗传缺陷是不同且不连锁的，并且常染色体以及X连锁基因控制未免疫小鼠的血清免疫球蛋白M。
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