LeVeen H H, Vujic I, d'Ovidio N G, Hutto R B
Ann Surg. 1984 Aug;200(2):212-23. doi: 10.1097/00000658-198408000-00016.
Electronic pressure testing of every LeVeen valve has practically eliminated mechanical malfunction as a cause of shunt failure. Nonetheless, failures do occur and in a series of 240 cases, early or late shunt failure occurred in 29 patients. Thirty-five additional cases of failures were either referred by other physicians over a period of 6 years or information and x-rays were accumulated by direct contact. Shunt failure becomes manifest by a sudden reaccumulation of ascites in patients with a previously functioning shunt. In immediate failure, the ascites may fail to disappear after surgery or reaccumulate if removed. Ideally, caval clotting should be first excluded by x-ray visualization of the superior vena prior to injection of the shunt with contrast agent. Shuntograms are done with fine-bore needles. The venous pressure is also measured. The entry of contrast into the vena cava without pooling indicates a patent venous limb. The contrast will empty from the venous tubing with forceful inspiration if the entire system is patent. The venous tube will not clear if the valve or peritoneal collecting tubes are blocked. Only the valve and collecting tube need then be replaced if contrast enters the cava but does not leave the venous tubing. Occluded valves must not be flushed to restore patency since inflammatory exudate and cellular debris are erroneously identified as "fibrin flecks." Histology and culture are mandatory. Immediate and early failure are often caused by malposition of the venous tubing. Malplacements can often be diagnosed simply by chest x-rays. Intraoperative injection of methylene blue into the venous tubing establishes a satisfactory washout prior to wound closure. Fresh clots in the vena cava can be dissolved by the slow injection of streptokinase into the venous tubing. Other patent veins are chosen for access. Patients having repeat surgery after clotting must be heparinized to prevent a similar recurrence. Flushing blood clots from the cava can be fatal.
对每个LeVeen瓣膜进行电子压力测试实际上已消除了机械故障作为分流失败的原因。尽管如此,分流失败仍会发生,在240例病例系列中,29例患者出现了早期或晚期分流失败。另外35例失败病例要么是在6年期间由其他医生转诊而来,要么是通过直接接触积累了相关信息和X光片。分流失败表现为先前功能正常的分流患者腹水突然再次积聚。在即刻失败的情况下,腹水可能在手术后未消失或在清除后再次积聚。理想情况下,在向分流器注射造影剂之前,应先通过上腔静脉的X光显影排除腔静脉凝血。用细针进行分流造影。同时测量静脉压力。造影剂进入腔静脉而不积聚表明静脉分支通畅。如果整个系统通畅,造影剂将在用力吸气时从静脉导管中排空。如果瓣膜或腹膜收集管堵塞,静脉导管将不会排空。如果造影剂进入腔静脉但未离开静脉导管,那么只需更换瓣膜和收集管。堵塞的瓣膜绝不能冲洗以恢复通畅,因为炎性渗出物和细胞碎片会被错误地识别为“纤维蛋白斑点”。必须进行组织学检查和培养。即刻和早期失败通常是由静脉导管位置不当引起的。位置不当通常仅凭胸部X光片就能诊断。术中向静脉导管内注射亚甲蓝可在伤口闭合前建立满意的冲洗效果。腔静脉内的新鲜血栓可通过向静脉导管内缓慢注射链激酶来溶解。选择其他通畅的静脉进行接入。凝血后进行再次手术的患者必须进行肝素化以防止类似复发。从腔静脉冲洗血凝块可能是致命的。