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Hydroperoxide effects on redox state of pyridine nucleotides and Ca2+ retention by mitochondria.

作者信息

Richter C

出版信息

Methods Enzymol. 1984;105:435-41. doi: 10.1016/s0076-6879(84)05061-8.

DOI:10.1016/s0076-6879(84)05061-8
PMID:6727679
Abstract
摘要

相似文献

1
Hydroperoxide effects on redox state of pyridine nucleotides and Ca2+ retention by mitochondria.
Methods Enzymol. 1984;105:435-41. doi: 10.1016/s0076-6879(84)05061-8.
2
Pyridine-nucleotide oxidation, Ca2+ cycling and membrane damage during tert-butyl hydroperoxide metabolism by rat-liver mitochondria.叔丁基过氧化氢在大鼠肝脏线粒体中代谢时的吡啶核苷酸氧化、钙离子循环及膜损伤
Eur J Biochem. 1984 Apr 2;140(1):1-6. doi: 10.1111/j.1432-1033.1984.tb08058.x.
3
Hydroperoxide-induced loss of pyridine nucleotides and release of calcium from rat liver mitochondria.过氧化氢诱导大鼠肝脏线粒体中吡啶核苷酸的丢失及钙的释放。
J Biol Chem. 1980 Oct 10;255(19):9325-30.
4
Hydroperoxides can modulate the redox state of pyridine nucleotides and the calcium balance in rat liver mitochondria.氢过氧化物可调节大鼠肝线粒体中吡啶核苷酸的氧化还原状态和钙平衡。
Proc Natl Acad Sci U S A. 1979 Sep;76(9):4340-4. doi: 10.1073/pnas.76.9.4340.
5
ATP prevents both hydroperoxide-induced hydrolysis of pyridine nucleotides and release of calcium in rat liver mitochondria.三磷酸腺苷可防止大鼠肝线粒体中氢过氧化物诱导的吡啶核苷酸水解及钙释放。
Eur J Biochem. 1981 Jul;117(2):361-7. doi: 10.1111/j.1432-1033.1981.tb06346.x.
6
Quantitative and mechanistic aspects of the hydroperoxide-induced release of Ca2+ from rat liver mitochondria.氢过氧化物诱导大鼠肝线粒体释放Ca2+的定量及机制方面
Eur J Biochem. 1985 Jun 18;149(3):633-9. doi: 10.1111/j.1432-1033.1985.tb08971.x.
7
Retention of Ca2+ by rat liver and rat heart mitochondria: effect of phosphate, Mg2+, and NAD(P) redox state.大鼠肝脏和大鼠心脏线粒体对Ca2+的潴留:磷酸盐、Mg2+和NAD(P)氧化还原状态的影响。
Arch Biochem Biophys. 1980 Oct 1;204(1):141-7. doi: 10.1016/0003-9861(80)90016-8.
8
The participation of NADP, the transmembrane potential and the energy-linked NAD(P) transhydrogenase in the process of Ca2+ efflux from rat liver mitochondria.烟酰胺腺嘌呤二核苷酸磷酸(NADP)、跨膜电位以及能量偶联的烟酰胺腺嘌呤二核苷酸(磷酸)转氢酶在大鼠肝脏线粒体钙离子外流过程中的作用。
Arch Biochem Biophys. 1987 Jan;252(1):171-8. doi: 10.1016/0003-9861(87)90021-x.
9
Hydroperoxide-stimulated release of calcium from rat liver and AS-30D hepatoma mitochondria.过氧化氢刺激大鼠肝脏和AS - 30D肝癌线粒体释放钙。
Cancer Res. 1986 Jul;46(7):3459-63.
10
t-Butylhydroperoxide-induced Ca2+ efflux from liver mitochondria in the presence of physiological concentrations of Mg2+ and ATP.在生理浓度的镁离子(Mg2+)和三磷酸腺苷(ATP)存在的情况下,叔丁基过氧化氢诱导肝线粒体中的钙离子(Ca2+)外流。
Biochim Biophys Acta. 1986 Jun 10;850(1):41-8. doi: 10.1016/0005-2728(86)90006-x.

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Frataxin activates mitochondrial energy conversion and oxidative phosphorylation.
铁调素激活线粒体能量转换和氧化磷酸化。
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Metabolic pathways for activation of the antiviral agent 9-(2-phosphonylmethoxyethyl)adenine in human lymphoid cells.人淋巴细胞中抗病毒药物9-(2-膦酰甲氧基乙基)腺嘌呤的代谢活化途径。
Antimicrob Agents Chemother. 1995 Oct;39(10):2304-8. doi: 10.1128/AAC.39.10.2304.
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Oxidative damage to mitochondria is mediated by the Ca(2+)-dependent inner-membrane permeability transition.线粒体的氧化损伤由钙离子依赖的内膜通透性转变介导。
Biochem J. 1993 Sep 15;294 ( Pt 3)(Pt 3):719-25. doi: 10.1042/bj2940719.
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The activity of xanthine oxidase in heart of pigs, guinea pigs, rabbits, rats, and humans.猪、豚鼠、兔、大鼠和人类心脏中黄嘌呤氧化酶的活性。
Basic Res Cardiol. 1987 Sep-Oct;82(5):486-92. doi: 10.1007/BF01907096.
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Mitochondrial calcium in hearts subjected to lipid peroxidation with contracture development.
Basic Res Cardiol. 1989 Nov-Dec;84(6):646-52. doi: 10.1007/BF01906949.