迟发型超敏反应（DTH）转移的I-A亚区控制 - Suppr

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超能文献

I-A subregion control of the transfer of delayed type hypersensitivity (DTH).

作者信息

Sandrin M S, McKenzie I F

出版信息

Immunogenetics. 1982;15(6):615-9. doi: 10.1007/BF00347057.

DOI:10.1007/BF00347057

PMID:6955277

Abstract

摘要

相似文献

I-A subregion control of the transfer of delayed type hypersensitivity (DTH).迟发型超敏反应（DTH）转移的I-A亚区控制

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[Genetic restriction of delayed-type hypersensitivity to tuberculin in mice].

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The defect in delayed-type hypersensitivity of young adult SJL mice is due to a lack of functional antigen-presenting cells.年轻成年SJL小鼠迟发型超敏反应的缺陷是由于缺乏功能性抗原呈递细胞。

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Immunogenetic analysis of tolerance induction in anti-alloantigen delayed type hypersensitivity responses by portal venous pre-inoculation with allogeneic cells.通过门静脉预先接种同种异体细胞对同种异体抗原迟发型超敏反应中耐受诱导的免疫遗传学分析。

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Alloantigens introduced into the anterior chamber of the eye induce systemic suppression of delayed hypersensitivity to third-party alloantigens through "linked recognition".引入眼房水的同种异体抗原通过“连锁识别”诱导对第三方同种异体抗原的迟发型超敏反应的全身抑制。

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[Regulation of the intensity of delayed-type hypersensitivity to sheep erythrocytes by the K-region of the major histocompatibility complex in mice].[小鼠主要组织相容性复合体K区对绵羊红细胞迟发型超敏反应强度的调节]

Biull Eksp Biol Med. 1981 Nov;92(11):577-8.

Hapten-specific T-cell responses to 4-hydroxy-3-nitrophenyl acetyl. I. Genetic control of delayed-type hypersensitivity by VH and I-A-region genes.对半抗原4-羟基-3-硝基苯乙酰的特异性T细胞应答。I. VH和I-A区基因对迟发型超敏反应的遗传控制。

J Exp Med. 1979 Jun 1;149(6):1336-48. doi: 10.1084/jem.149.6.1336.

本文引用的文献

Two T-cell populations mediate delayed-type hypersensitivity to murine influenza virus infection.两种T细胞群体介导对小鼠流感病毒感染的迟发型超敏反应。

Scand J Immunol. 1980;12(6):481-7. doi: 10.1111/j.1365-3083.1980.tb00094.x.

Ir gene function in an I-A subregion mutant B6.C-H-2bm12.I-A亚区突变体B6.C-H-2bm12中的Ir基因功能

J Exp Med. 1981 Feb 1;153(2):464-9. doi: 10.1084/jem.153.2.464.

Serological analysis of B6.C-H-2bm12 (I-A mutant).

Eur J Immunol. 1982 Mar;12(3):205-9. doi: 10.1002/eji.1830120307.

Cytotoxic T lymphocytes generated across and I-Ab mutant difference are directed against a molecule bearing Ia antigens.跨越I-Ab突变差异产生的细胞毒性T淋巴细胞针对携带Ia抗原的分子。

Eur J Immunol. 1981 Mar;11(3):258-65. doi: 10.1002/eji.1830110317.

Analysis of histocompatibility requirements for proliferative and helper T cell activity. T cell populations depleted of alloreactive cells by negative selection.增殖性和辅助性T细胞活性的组织相容性需求分析。通过阴性选择去除同种反应性细胞的T细胞群体。

J Exp Med. 1980 Nov 1;152(5):1311-28. doi: 10.1084/jem.152.5.1311.

Delayed-type hypersensitivity induced by antigen-pulsed, bone marrow-derived macrophages.

Eur J Immunol. 1980 Mar;10(3):165-70. doi: 10.1002/eji.1830100302.

In vitro cell-mediated immune responses to the male specific(H-Y) antigen in mice.小鼠对雄性特异性（H-Y）抗原的体外细胞介导免疫反应。

J Exp Med. 1975 Nov 1;142(5):1108-20. doi: 10.1084/jem.142.5.1108.

On the role of the H-2 histocompatibility complex in determining the specificity of cytotoxic effector cells sensitized against syngeneic trinitrophenyl-modified targets.关于H-2组织相容性复合体在确定针对同基因三硝基苯基修饰靶标的细胞毒性效应细胞特异性中的作用。

J Exp Med. 1975 Aug 1;142(2):403-18. doi: 10.1084/jem.142.2.403.

B6.C-H-2bm12. A new H-2 mutation in the I region in the mouse.B6.C-H-2bm12。小鼠I区的一种新的H-2突变。

J Exp Med. 1979 Dec 1;150(6):1323-38. doi: 10.1084/jem.150.6.1323.

The major histocompatibility complex determines susceptibility to cytotoxic T cells directed against minor histocompatibility antigens.主要组织相容性复合体决定了针对次要组织相容性抗原的细胞毒性T细胞的易感性。

J Exp Med. 1975 Dec 1;142(6):1349-64. doi: 10.1084/jem.142.6.1349.

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