Jouvet M
Department of Experimental Medicine, Claude Bernard University, Lyon, France.
Sleep. 1994 Dec;17(8 Suppl):S77-83. doi: 10.1093/sleep/17.suppl_8.s77.
A review of the effects of brain-stem transections, carbachol microinjections and lesions does not show that the pons is necessary and sufficient for paradoxical sleep (PS) generation. The different theories that explain the mechanisms of PS rebound are summarized. In some circumstances, PS rebound depends upon the stressful quality of PS deprivation. PS appears to necessitate oxidative metabolism of glucose and, in certain situations, PS may be increased by hyperoxia.
对脑干横切、卡巴胆碱微量注射及损伤的影响进行回顾后发现，脑桥对于异相睡眠（PS）的产生并非必要且充分条件。文中总结了解释PS反跳机制的不同理论。在某些情况下，PS反跳取决于PS剥夺的应激性质。PS似乎需要葡萄糖的氧化代谢，并且在某些情况下，高氧可能会增加PS。
