Oral tolerance.
作者信息
Weiner H L
机构信息
Center for Neurologic Diseases, Brigham and Women's Hospital, Boston, MA 02115.
出版信息
Proc Natl Acad Sci U S A. 1994 Nov 8;91(23):10762-5. doi: 10.1073/pnas.91.23.10762.
Abstract
摘要
相似文献
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Oral tolerance.口服耐受
Proc Natl Acad Sci U S A. 1994 Nov 8;91(23):10762-5. doi: 10.1073/pnas.91.23.10762.
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Manipulation of Th responses by oral tolerance.通过口服耐受来调控Th反应。
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The in vitro production of cytokines by mucosal lymphocytes immunized by oral administration of keyhole limpet hemocyanin using cholera toxin as an adjuvant.以霍乱毒素作为佐剂，通过口服钥孔戚血蓝蛋白免疫的黏膜淋巴细胞在体外产生细胞因子的情况。
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Gastrointestinal lesions associated with spondyloarthropathies.与脊柱关节病相关的胃肠道病变。
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Orally tolerant CD4 T cells respond poorly to antigenic stimulation but strongly to direct stimulation of intracellular signaling pathways.口服耐受的 CD4 T 细胞对抗原刺激的反应较差，但对细胞内信号通路的直接刺激反应强烈。
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本文引用的文献
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Oral, but not intravenous, alloantigen prevents accelerated allograft rejection by selective intragraft Th2 cell activation.口服而非静脉注射同种异体抗原可通过选择性激活移植组织内的Th2细胞来预防移植器官的加速排斥反应。
Transplantation. 1993 May;55(5):1112-8. doi: 10.1097/00007890-199305000-00034.
2
Effects of oral administration of type II collagen on rheumatoid arthritis.口服II型胶原蛋白对类风湿性关节炎的影响。
Science. 1993 Sep 24;261(5129):1727-30. doi: 10.1126/science.8378772.
3
Helper T cell subsets for immunoglobulin A responses: oral immunization with tetanus toxoid and cholera toxin as adjuvant selectively induces Th2 cells in mucosa associated tissues.参与免疫球蛋白A应答的辅助性T细胞亚群：以破伤风类毒素和霍乱毒素作为佐剂进行口服免疫，可在黏膜相关组织中选择性诱导Th2细胞。
J Exp Med. 1993 Oct 1;178(4):1309-20. doi: 10.1084/jem.178.4.1309.
4
Cholera toxin adjuvant greatly promotes antigen priming of T cells.霍乱毒素佐剂能极大地促进T细胞对抗原的致敏。
Eur J Immunol. 1993 Sep;23(9):2136-43. doi: 10.1002/eji.1830230914.
5
Immune response to glutamic acid decarboxylase correlates with insulitis in non-obese diabetic mice.对谷氨酸脱羧酶的免疫反应与非肥胖糖尿病小鼠的胰岛炎相关。
Nature. 1993 Nov 4;366(6450):72-5. doi: 10.1038/366072a0.
6
Oral tolerance in humans. T cell but not B cell tolerance after antigen feeding.人类的口服耐受。抗原喂食后T细胞而非B细胞的耐受。
J Immunol. 1994 May 1;152(9):4663-70.
7
Induction of anergy or active suppression following oral tolerance is determined by antigen dosage.口服耐受后无反应性或主动抑制的诱导取决于抗原剂量。
Proc Natl Acad Sci U S A. 1994 Jul 5;91(14):6688-92. doi: 10.1073/pnas.91.14.6688.
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Oral tolerance: immunologic mechanisms and treatment of animal and human organ-specific autoimmune diseases by oral administration of autoantigens.口服耐受：通过口服自身抗原对动物和人类器官特异性自身免疫性疾病的免疫机制及治疗
Annu Rev Immunol. 1994;12:809-37. doi: 10.1146/annurev.iy.12.040194.004113.
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Spontaneous loss of T-cell tolerance to glutamic acid decarboxylase in murine insulin-dependent diabetes.小鼠胰岛素依赖型糖尿病中T细胞对谷氨酸脱羧酶的自发耐受性丧失。
Nature. 1993 Nov 4;366(6450):69-72. doi: 10.1038/366069a0.
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