Hirschel B J, Benusiglio L N, Favre H, Chatelanat F, Humair L, Zubler R H, Cruchaud A
Clin Nephrol. 1977 Sep;8(3):404-9.
A 45-year old developed membranoproliferative GN seven years after acute hepatitis. He was found to be a chronic carrier of HBsAg, and glomeruli contained granular deposits of immunoglobulins (Ig), complement (C) and HBsAg. Six months later, HN persisted, but HBsAg has disappeared from glomeruli; Ig and C were still present. It was concluded that GN was probably due to a hepatitis B associated antigen, but not necessarily to HBsAg.
一名45岁男性在急性肝炎7年后发展为膜增生性肾小球肾炎(GN)。他被发现是乙肝表面抗原(HBsAg)的慢性携带者,肾小球中含有免疫球蛋白(Ig)、补体(C)和HBsAg的颗粒状沉积物。6个月后,GN持续存在,但HBsAg已从肾小球中消失;Ig和C仍然存在。结论是,GN可能是由一种与乙肝相关的抗原引起的,但不一定是由HBsAg引起的。