肠道病毒70结合需要衰变加速因子的短共有重复结构域1。
Short consensus repeat domain 1 of decay-accelerating factor is required for enterovirus 70 binding.
作者信息
Karnauchow T M, Dawe S, Lublin D M, Dimock K
机构信息
Department of Biochemistry, Microbiology and Immunology, University of Ottawa, Ottawa, Ontario, Canada K1H 8M5.
出版信息
J Virol. 1998 Nov;72(11):9380-3. doi: 10.1128/JVI.72.11.9380-9383.1998.
Abstract
Enterovirus 70 (EV70), like several other human enteroviruses, can utilize decay-accelerating factor (DAF [CD55]) as an attachment protein. Using chimeric molecules composed of different combinations of the short consensus repeat domains (SCRs) of DAF and membrane cofactor protein (CD46), we show that sequences in SCR1 of DAF are essential for EV70 binding. Of the human enteroviruses that can bind to DAF, only EV70 and coxsackievirus A21 require sequences in SCR1 for this interaction.
摘要
肠道病毒70型（EV70）与其他几种人类肠道病毒一样，可利用衰变加速因子（DAF [CD55]）作为附着蛋白。我们使用由DAF和膜辅因子蛋白（CD46）的短共有重复序列结构域（SCR）的不同组合组成的嵌合分子，证明DAF的SCR1中的序列对于EV70结合至关重要。在可与DAF结合的人类肠道病毒中，只有EV70和柯萨奇病毒A21型需要SCR1中的序列进行这种相互作用。
相似文献
1
Short consensus repeat domain 1 of decay-accelerating factor is required for enterovirus 70 binding.肠道病毒70结合需要衰变加速因子的短共有重复结构域1。
J Virol. 1998 Nov;72(11):9380-3. doi: 10.1128/JVI.72.11.9380-9383.1998.
2
The HeLa cell receptor for enterovirus 70 is decay-accelerating factor (CD55).肠道病毒70的海拉细胞受体是衰变加速因子（CD55）。
J Virol. 1996 Aug;70(8):5143-52. doi: 10.1128/JVI.70.8.5143-5152.1996.
3
Mapping the binding domains on decay accelerating factor (DAF) for haemagglutinating enteroviruses: implications for the evolution of a DAF-binding phenotype.绘制血凝性肠道病毒在衰变加速因子（DAF）上的结合域：对DAF结合表型进化的启示
J Gen Virol. 1999 Dec;80 ( Pt 12):3145-3152. doi: 10.1099/0022-1317-80-12-3145.
4
Measles virus and C3 binding sites are distinct on membrane cofactor protein (CD46).麻疹病毒和C3结合位点在膜辅因子蛋白（CD46）上是不同的。
Proc Natl Acad Sci U S A. 1995 Mar 14;92(6):2303-7. doi: 10.1073/pnas.92.6.2303.
5
Chimeric CD46/DAF molecules reveal a cryptic functional role for SCR1 of DAF in regulating complement activation.嵌合CD46/衰变加速因子（DAF）分子揭示了DAF的SCR1在调节补体激活中的隐秘功能作用。
Mol Immunol. 2000 Aug-Sep;37(12-13):687-96. doi: 10.1016/s0161-5890(01)00002-5.
6
Coxsackievirus A21 binds to decay-accelerating factor but requires intercellular adhesion molecule 1 for cell entry.柯萨奇病毒A21与衰变加速因子结合，但细胞进入需要细胞间黏附分子1。
J Virol. 1997 Jun;71(6):4736-43. doi: 10.1128/JVI.71.6.4736-4743.1997.
7
Coxsackievirus B3 adapted to growth in RD cells binds to decay-accelerating factor (CD55).适应在RD细胞中生长的柯萨奇病毒B3与衰变加速因子（CD55）结合。
J Virol. 1995 Mar;69(3):1903-6. doi: 10.1128/JVI.69.3.1903-1906.1995.
8
Binding to decay-accelerating factor is not required for infection of human leukocyte cell lines by enterovirus 70.肠道病毒70感染人白细胞细胞系并不需要与衰变加速因子结合。
J Virol. 2004 Mar;78(6):2674-81. doi: 10.1128/jvi.78.6.2674-2681.2004.
9
Coxsackie B viruses that use human DAF as a receptor infect pig cells via pig CAR and do not use pig DAF.利用人类衰变加速因子（DAF）作为受体的柯萨奇B病毒通过猪柯萨奇病毒和腺病毒受体（CAR）感染猪细胞，而不利用猪DAF。
J Gen Virol. 2002 Jan;83(Pt 1):45-52. doi: 10.1099/0022-1317-83-1-45.
10
Interaction between echovirus 7 and its receptor, decay-accelerating factor (CD55): evidence for a secondary cellular factor in A-particle formation.埃可病毒7与其受体衰变加速因子（CD55）之间的相互作用：A颗粒形成中二级细胞因子的证据。
J Virol. 1997 Dec;71(12):9306-12. doi: 10.1128/JVI.71.12.9306-9312.1997.
引用本文的文献
1
Structure of Human Enterovirus 70 and Its Inhibition by Capsid-Binding Compounds.人肠道病毒 70 结构及其衣壳结合化合物的抑制作用。
J Virol. 2022 Sep 14;96(17):e0060422. doi: 10.1128/jvi.00604-22. Epub 2022 Aug 8.
2
: A Small but Versatile Species.一个小型但用途广泛的物种。
Microorganisms. 2021 Aug 17;9(8):1758. doi: 10.3390/microorganisms9081758.
3
In the Crosshairs: RNA Viruses OR Complement?在瞄准镜中：RNA 病毒还是补体？
Front Immunol. 2020 Sep 29;11:573583. doi: 10.3389/fimmu.2020.573583. eCollection 2020.
4
Genetic and phenotypic characterization of recently discovered enterovirus D type 111.最近发现的肠道病毒 D 型 111 的遗传和表型特征。
PLoS Negl Trop Dis. 2019 Oct 17;13(10):e0007797. doi: 10.1371/journal.pntd.0007797. eCollection 2019 Oct.
5
Complement therapeutics meets nanomedicine: overcoming human complement activation and leukocyte uptake of nanomedicines with soluble domains of CD55.补体治疗与纳米医学相遇：用 CD55 的可溶性结构域克服纳米药物的人补体激活和白细胞摄取。
J Control Release. 2019 May 28;302:181-189. doi: 10.1016/j.jconrel.2019.04.009. Epub 2019 Apr 8.
6
Complement Evasion Strategies of Viruses: An Overview.病毒的补体逃避策略：概述
Front Microbiol. 2017 Jun 16;8:1117. doi: 10.3389/fmicb.2017.01117. eCollection 2017.
7
Blood Groups in Infection and Host Susceptibility.感染与宿主易感性中的血型
Clin Microbiol Rev. 2015 Jul;28(3):801-70. doi: 10.1128/CMR.00109-14.
8
A rapid and efficient method for studies of virus interaction at the host cell surface using enteroviruses and real-time PCR.利用肠道病毒和实时 PCR 技术研究病毒在宿主细胞表面相互作用的快速高效方法。
Virol J. 2009 Dec 7;6:217. doi: 10.1186/1743-422X-6-217.
9
Interaction of decay-accelerating factor with coxsackievirus B3.衰变加速因子与柯萨奇病毒B3的相互作用
J Virol. 2007 Dec;81(23):12927-35. doi: 10.1128/JVI.00931-07. Epub 2007 Sep 5.
10
Enterovirus 70 receptor utilization is controlled by capsid residues that also regulate host range and cytopathogenicity.肠道病毒70型受体的利用受衣壳残基控制，这些残基也调节宿主范围和细胞致病性。
J Virol. 2007 Aug;81(16):8648-55. doi: 10.1128/JVI.01569-06. Epub 2007 May 30.
本文引用的文献
1
Characterization of echoviruses that bind decay accelerating factor (CD55): evidence that some haemagglutinating strains use more than one cellular receptor.与衰变加速因子（CD55）结合的艾柯病毒的特性：一些血凝性毒株使用不止一种细胞受体的证据。
J Gen Virol. 1998 Jul;79 ( Pt 7):1707-13. doi: 10.1099/0022-1317-79-7-1707.
2
Role for beta2-microglobulin in echovirus infection of rhabdomyosarcoma cells.β2-微球蛋白在横纹肌肉瘤细胞肠道病毒感染中的作用。
J Virol. 1998 Jul;72(7):5360-5. doi: 10.1128/JVI.72.7.5360-5365.1998.
3
Cardiovirulent coxsackieviruses and the decay-accelerating factor (CD55) receptor.具有心脏毒性的柯萨奇病毒与衰变加速因子（CD55）受体
Virology. 1998 May 10;244(2):302-14. doi: 10.1006/viro.1998.9122.
4
The murine CAR homolog is a receptor for coxsackie B viruses and adenoviruses.小鼠CAR同源物是柯萨奇B病毒和腺病毒的受体。
J Virol. 1998 Jan;72(1):415-9. doi: 10.1128/JVI.72.1.415-419.1998.
5
A decay-accelerating factor-binding strain of coxsackievirus B3 requires the coxsackievirus-adenovirus receptor protein to mediate lytic infection of rhabdomyosarcoma cells.柯萨奇病毒B3的衰变加速因子结合株需要柯萨奇病毒-腺病毒受体蛋白来介导横纹肌肉瘤细胞的裂解感染。
J Virol. 1997 Dec;71(12):9844-8. doi: 10.1128/JVI.71.12.9844-9848.1997.
6
Interaction between echovirus 7 and its receptor, decay-accelerating factor (CD55): evidence for a secondary cellular factor in A-particle formation.埃可病毒7与其受体衰变加速因子（CD55）之间的相互作用：A颗粒形成中二级细胞因子的证据。
J Virol. 1997 Dec;71(12):9306-12. doi: 10.1128/JVI.71.12.9306-9312.1997.
7
Coxsackievirus A21 binds to decay-accelerating factor but requires intercellular adhesion molecule 1 for cell entry.柯萨奇病毒A21与衰变加速因子结合，但细胞进入需要细胞间黏附分子1。
J Virol. 1997 Jun;71(6):4736-43. doi: 10.1128/JVI.71.6.4736-4743.1997.
8
HCAR and MCAR: the human and mouse cellular receptors for subgroup C adenoviruses and group B coxsackieviruses.HCAR和MCAR：C亚群腺病毒和B组柯萨奇病毒的人类和小鼠细胞受体。
Proc Natl Acad Sci U S A. 1997 Apr 1;94(7):3352-6. doi: 10.1073/pnas.94.7.3352.
9
Clinical coxsackievirus B isolates differ from laboratory strains in their interaction with two cell surface receptors.
J Infect Dis. 1997 Mar;175(3):697-700. doi: 10.1093/infdis/175.3.697.
10
Isolation of a common receptor for Coxsackie B viruses and adenoviruses 2 and 5.柯萨奇B病毒以及腺病毒2型和5型共同受体的分离
Science. 1997 Feb 28;275(5304):1320-3. doi: 10.1126/science.275.5304.1320.
Zotero 插件