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Creatine kinase: an enzyme with a central role in cellular energy metabolism.

作者信息

Wallimann T, Dolder M, Schlattner U, Eder M, Hornemann T, Kraft T, Stolz M

机构信息

Institute of Cell Biology, ETH-Hönggerberg, Zürich, Switzerland.

出版信息

MAGMA. 1998 Sep;6(2-3):116-9. doi: 10.1007/BF02660927.

DOI:10.1007/BF02660927
PMID:9803379
Abstract
摘要

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1
Creatine kinase: an enzyme with a central role in cellular energy metabolism.肌酸激酶：一种在细胞能量代谢中起核心作用的酶。
MAGMA. 1998 Sep;6(2-3):116-9. doi: 10.1007/BF02660927.
2
Energetic basis of diastolic dysfunction.舒张功能障碍的能量基础。
MAGMA. 1998 Sep;6(2-3):129-31. doi: 10.1007/BF02660932.
3
Effects of ischemia on skeletal muscle energy metabolism in mice lacking creatine kinase monitored by in vivo 31P nuclear magnetic resonance spectroscopy.通过体内31P核磁共振波谱监测缺血对缺乏肌酸激酶的小鼠骨骼肌能量代谢的影响。
NMR Biomed. 1999 Oct;12(6):327-34. doi: 10.1002/(sici)1099-1492(199910)12:6<327::aid-nbm570>3.0.co;2-9.
4
[Creatine kinase system and muscle energy metabolism].[肌酸激酶系统与肌肉能量代谢]
Zh Obshch Biol. 1981 Jul-Aug;42(4):586-96.
5
Impaired cardiac energetics in mice lacking muscle-specific isoenzymes of creatine kinase.缺乏肌酸激酶肌肉特异性同工酶的小鼠心脏能量代谢受损。
Circ Res. 1998 May 4;82(8):898-907. doi: 10.1161/01.res.82.8.898.
6
Impaired intracellular energetic communication in muscles from creatine kinase and adenylate kinase (M-CK/AK1) double knock-out mice.肌酸激酶和腺苷酸激酶（M-CK/AK1）双敲除小鼠肌肉中细胞内能量传递受损。
J Biol Chem. 2003 Aug 15;278(33):30441-9. doi: 10.1074/jbc.M303150200. Epub 2003 May 1.
7
Mathematical model of energy transfer in hearts with inhibited or ablated creatine kinase system.肌酸激酶系统受抑制或被切除的心脏中能量转移的数学模型。
MAGMA. 1998 Sep;6(2-3):124-5. doi: 10.1007/BF02660930.
8
Creatine kinase knockout mice--what is the phenotype: heart.肌酸激酶基因敲除小鼠——表型是什么：心脏方面。
MAGMA. 1998 Sep;6(2-3):120-1.
9
Creatine kinase knockout mice--what is the phenotype: skeletal muscle.肌酸激酶基因敲除小鼠——表型是什么：骨骼肌。
MAGMA. 1998 Sep;6(2-3):122-3. doi: 10.1007/BF02660929.
10
31P NMR studies of creatine kinase flux in M-creatine kinase-deficient mouse heart.对肌酸激酶缺乏的小鼠心脏中肌酸激酶通量的31P核磁共振研究。
Am J Physiol. 1998 Oct;275(4):H1191-9. doi: 10.1152/ajpheart.1998.275.4.H1191.

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Transcriptomics and proteomics provide insights into the adaptative strategies of Tibetan naked carps (Gymnocypris przewalskii) to saline-alkaline variations.转录组学和蛋白质组学为青海湖裸鲤适应盐碱变化的策略提供了见解。
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本文引用的文献

1
Oligomeric state and membrane binding behaviour of creatine kinase isoenzymes: implications for cellular function and mitochondrial structure.肌酸激酶同工酶的寡聚状态及膜结合行为：对细胞功能和线粒体结构的影响
Mol Cell Biochem. 1998 Jul;184(1-2):141-51.
2
Functional aspects of the X-ray structure of mitochondrial creatine kinase: a molecular physiology approach.线粒体肌酸激酶X射线结构的功能方面：一种分子生理学方法。
Mol Cell Biochem. 1998 Jul;184(1-2):125-40.
3
Mitochondrial creatine kinase is a prime target of peroxynitrite-induced modification and inactivation.
肌酸作为抗惊厥剂的潜在作用：来自临床前研究的证据。
Front Neurosci. 2023 Jun 29;17:1201971. doi: 10.3389/fnins.2023.1201971. eCollection 2023.
4
Low Tissue Creatine: A Therapeutic Target in Clinical Nutrition.低组织肌酸：临床营养中的治疗靶点。
Nutrients. 2022 Mar 15;14(6):1230. doi: 10.3390/nu14061230.
5
Effects of Creatine Supplementation on Brain Function and Health.肌酸补充对大脑功能和健康的影响。
Nutrients. 2022 Feb 22;14(5):921. doi: 10.3390/nu14050921.
6
Expression patterns of regulatory lncRNAs and miRNAs in muscular atrophy models induced by starvation in vitro and in vivo.体外和体内饥饿诱导的肌肉萎缩模型中调节 lncRNAs 和 miRNAs 的表达模式。
Mol Med Rep. 2019 Nov;20(5):4175-4185. doi: 10.3892/mmr.2019.10661. Epub 2019 Sep 10.
7
Creatine supplementation improves neural progenitor cell survival in Huntington's disease.补充肌酸可改善亨廷顿舞蹈病中神经祖细胞的存活率。
Brain Circ. 2016 Jul-Sep;2(3):133-137. doi: 10.4103/2394-8108.192519. Epub 2016 Oct 18.
8
Disruption of Energy Transfer and Redox Status by Sulfite in Hippocampus, Striatum, and Cerebellum of Developing Rats.亚硫酸盐对发育中大鼠海马、纹状体和小脑中能量传递和氧化还原状态的破坏作用
Neurotox Res. 2017 Aug;32(2):264-275. doi: 10.1007/s12640-017-9732-y. Epub 2017 Apr 17.
9
Mechanistic effects of amino acids and glucose in a novel glutaric aciduria type 1 cell model.新型1型戊二酸血症细胞模型中氨基酸和葡萄糖的作用机制
PLoS One. 2014 Oct 15;9(10):e110181. doi: 10.1371/journal.pone.0110181. eCollection 2014.
10
Coenzyme Q10 effects on creatine kinase activity and mood in geriatric bipolar depression.辅酶 Q10 对老年双相抑郁症患者肌酸激酶活性和情绪的影响。
J Geriatr Psychiatry Neurol. 2012 Mar;25(1):43-50. doi: 10.1177/0891988712436688.
线粒体肌酸激酶是过氧亚硝酸盐诱导修饰和失活的主要靶点。
J Biol Chem. 1998 Jul 3;273(27):16694-9. doi: 10.1074/jbc.273.27.16694.
4
Myofibrillar interaction of cytosolic creatine kinase (CK) isoenzymes: allocation of N-terminal binding epitope in MM-CK and BB-CK.胞质肌酸激酶（CK）同工酶的肌原纤维相互作用：MM-CK和BB-CK中N端结合表位的定位
J Cell Sci. 1998 May;111 ( Pt 9):1207-16. doi: 10.1242/jcs.111.9.1207.
5
Does muscle creatine phosphokinase have access to the total pool of phosphocreatine plus creatine?肌肉中的肌酸磷酸激酶能否接触到磷酸肌酸和肌酸的总量？
Am J Physiol. 1998 Mar;274(3):R868-72. doi: 10.1152/ajpregu.1998.274.3.R868.
6
Dual regulation of the AMP-activated protein kinase provides a novel mechanism for the control of creatine kinase in skeletal muscle.AMP 激活的蛋白激酶的双重调节为骨骼肌中肌酸激酶的调控提供了一种新机制。
EMBO J. 1998 Mar 16;17(6):1688-99. doi: 10.1093/emboj/17.6.1688.
7
The role of creatine kinase in inhibition of mitochondrial permeability transition.肌酸激酶在抑制线粒体通透性转换中的作用。
FEBS Lett. 1997 Sep 8;414(2):253-7. doi: 10.1016/s0014-5793(97)01045-4.
8
Kinetics of ATP to ADP beta-phosphoryl conversion in contracting skeletal muscle by in vivo 31P NMR magnetization transfer.通过体内31P NMR磁化转移研究收缩骨骼肌中ATP向ADPβ-磷酸化转化的动力学。
NMR Biomed. 1997 Apr;10(2):67-72. doi: 10.1002/(sici)1099-1492(199704)10:2<67::aid-nbm451>3.0.co;2-d.
9
Activation of sea-urchin sperm motility is accompanied by an increase in the creatine kinase exchange flux.海胆精子活力的激活伴随着肌酸激酶交换通量的增加。
Biochem J. 1997 Jul 15;325 ( Pt 2)(Pt 2):411-6. doi: 10.1042/bj3250411.
10
Crystalline mitochondrial inclusion bodies isolated from creatine depleted rat soleus muscle.从肌酸耗竭的大鼠比目鱼肌中分离出的结晶状线粒体包涵体。
J Cell Sci. 1997 Jun;110 ( Pt 12):1403-11. doi: 10.1242/jcs.110.12.1403.