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Capacity of Mycobacterium avium isolates to grow well or poorly in murine macrophages resides in their ability to induce secretion of tumor necrosis factor.鸟分枝杆菌分离株在小鼠巨噬细胞中生长良好或不良的能力取决于它们诱导肿瘤坏死因子分泌的能力。
Infect Immun. 1992 Oct;60(10):4410-3. doi: 10.1128/iai.60.10.4410-4413.1992.
2
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Activation of the mitogen-activated protein kinase signaling pathway is instrumental in determining the ability of Mycobacterium avium to grow in murine macrophages.丝裂原活化蛋白激酶信号通路的激活有助于确定鸟分枝杆菌在小鼠巨噬细胞中生长的能力。
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Elevated mitogen-activated protein kinase signalling and increased macrophage activation in cells infected with a glycopeptidolipid-deficient Mycobacterium avium.在感染糖脂缺陷型鸟分枝杆菌的细胞中,丝裂原活化蛋白激酶信号传导增强,巨噬细胞活化增加。
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7
Tumour necrosis factor-alpha (TNF-alpha) in the host resistance to mycobacteria of distinct virulence.肿瘤坏死因子-α(TNF-α)在宿主对不同毒力分枝杆菌的抵抗力中的作用。
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8
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Tumor necrosis factor and granulocyte macrophage-colony stimulating factor stimulate human macrophages to restrict growth of virulent Mycobacterium avium and to kill avirulent M. avium: killing effector mechanism depends on the generation of reactive nitrogen intermediates.肿瘤坏死因子和粒细胞巨噬细胞集落刺激因子刺激人类巨噬细胞限制有毒力的鸟分枝杆菌生长并杀死无毒力的鸟分枝杆菌:杀伤效应机制取决于活性氮中间体的产生。
J Leukoc Biol. 1991 Apr;49(4):380-7. doi: 10.1002/jlb.49.4.380.
10
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3
Host response to nontuberculous mycobacterial infections of current clinical importance.宿主对当前临床重要的非结核分枝杆菌感染的反应。
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4
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Control of mycobacterial replication in human macrophages: roles of extracellular signal-regulated kinases 1 and 2 and p38 mitogen-activated protein kinase pathways.人巨噬细胞中分枝杆菌复制的调控:细胞外信号调节激酶1和2以及p38丝裂原活化蛋白激酶途径的作用
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Minor role played by type I tumour necrosis factor receptor in the control of Mycobacterium avium proliferation in infected mice.I型肿瘤坏死因子受体在受感染小鼠中对鸟分枝杆菌增殖控制中的次要作用。
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Bacteremia due to Mycobacterium avium-intracellulare in the acquired immunodeficiency syndrome.获得性免疫缺陷综合征中鸟分枝杆菌-胞内分枝杆菌所致的菌血症
Ann Intern Med. 1983 Dec;99(6):782-5. doi: 10.7326/0003-4819-99-6-782.
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Characterization of the carbohydrate fragments obtained from Saccharomyces cerevisiae mannan by alkaline degradation.通过碱性降解对酿酒酵母甘露聚糖所得碳水化合物片段的表征。
J Biol Chem. 1974 Dec 10;249(23):7679-84.
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Comparison of 15 laboratory and patient-derived strains of Mycobacterium avium for ability to infect and multiply in cultured human macrophages.对15株实验室菌株和患者来源的鸟分枝杆菌菌株在培养的人巨噬细胞中感染和繁殖能力的比较。
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Mycobacterium avium complex infection.鸟分枝杆菌复合群感染
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Interferon-gamma and tumor necrosis factor induce the L-arginine-dependent cytotoxic effector mechanism in murine macrophages.干扰素-γ和肿瘤坏死因子诱导小鼠巨噬细胞中依赖L-精氨酸的细胞毒性效应机制。
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The epidemiology of disseminated nontuberculous mycobacterial infection in the acquired immunodeficiency syndrome (AIDS).获得性免疫缺陷综合征(艾滋病)中播散性非结核分枝杆菌感染的流行病学
Am Rev Respir Dis. 1989 Jan;139(1):4-7. doi: 10.1164/ajrccm/139.1.4.
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Mycobacteria and AIDS mortality.
Am Rev Respir Dis. 1989 Jan;139(1):1-3. doi: 10.1164/ajrccm/139.1.1.
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Tumor necrosis factor, alone or in combination with IL-2, but not IFN-gamma, is associated with macrophage killing of Mycobacterium avium complex.肿瘤坏死因子单独或与白细胞介素-2联合使用时(而非与γ干扰素联合使用),与巨噬细胞杀灭鸟分枝杆菌复合体有关。
J Immunol. 1988 May 1;140(9):3006-13.
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The inducing role of tumor necrosis factor in the development of bactericidal granulomas during BCG infection.卡介苗感染期间肿瘤坏死因子在杀菌性肉芽肿形成中的诱导作用。
Cell. 1989 Mar 10;56(5):731-40. doi: 10.1016/0092-8674(89)90676-4.
10
Treatment of experimental disseminated Mycobacterium avium complex infection in mice with recombinant IL-2 and tumor necrosis factor.用重组白细胞介素-2和肿瘤坏死因子治疗小鼠实验性播散性鸟分枝杆菌复合群感染
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鸟分枝杆菌分离株在小鼠巨噬细胞中生长良好或不良的能力取决于它们诱导肿瘤坏死因子分泌的能力。

Capacity of Mycobacterium avium isolates to grow well or poorly in murine macrophages resides in their ability to induce secretion of tumor necrosis factor.

作者信息

Furney S K, Skinner P S, Roberts A D, Appelberg R, Orme I M

机构信息

Department of Microbiology, Colorado State University, Fort Collins 80523.

出版信息

Infect Immun. 1992 Oct;60(10):4410-3. doi: 10.1128/iai.60.10.4410-4413.1992.

DOI:10.1128/iai.60.10.4410-4413.1992
PMID:1398951
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC257480/
Abstract

The results of this study show that clinical isolates of Mycobacterium avium fall into two categories in terms of their capacity to grow within murine bone marrow-derived macrophage cultures: those that grow progressively and those that are incapable of growing within such cells. Members of the first category were invariably of the smooth-transparent colonial type, while most of the second were of the smooth-doomed type. In addition, this paper shows that although all isolates induced tumor necrosis factor (TNF) secretion by host cells to some extent, this production was always delayed in isolates that subsequently grew well in the host cells. This observation, coupled with the demonstration that the growth of the latter isolates was inhibited by the exogenous addition of TNF, leads us to hypothesize that the ability of a given isolate to somehow avoid host macrophage TNF production early during the course of the infection is a key factor in the pathogenesis of the disease.

摘要

本研究结果表明,就鸟分枝杆菌临床分离株在鼠源骨髓巨噬细胞培养物中的生长能力而言,可分为两类:一类能持续生长,另一类无法在此类细胞中生长。第一类成员菌落类型始终为光滑透明型,而第二类大多数为光滑粗糙型。此外,本文表明,尽管所有分离株均能在一定程度上诱导宿主细胞分泌肿瘤坏死因子(TNF),但在随后能在宿主细胞中良好生长的分离株中,TNF的产生总是延迟的。这一观察结果,再加上已证明外源性添加TNF可抑制后一类分离株的生长,使我们推测,特定分离株在感染过程早期以某种方式避免宿主巨噬细胞产生TNF的能力是该疾病发病机制中的一个关键因素。