The Mus81 solution to resolution: generating meiotic crossovers without Holliday junctions.
作者信息
Hollingsworth Nancy M, Brill Steven J
机构信息
Department of Biochemistry and Cell Biology, Institute for Cell and Developmental Biology, State University of New York at Stony Brook, Stony Brook, NY 11794-5215, USA.
出版信息
Genes Dev. 2004 Jan 15;18(2):117-25. doi: 10.1101/gad.1165904.
Abstract
摘要
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The Mus81 solution to resolution: generating meiotic crossovers without Holliday junctions.Mus81解决重组问题的方式：在没有霍利迪连接体的情况下产生减数分裂交叉。
Genes Dev. 2004 Jan 15;18(2):117-25. doi: 10.1101/gad.1165904.
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本文引用的文献
1
Fission yeast Mus81.Eme1 Holliday junction resolvase is required for meiotic crossing over but not for gene conversion.裂殖酵母Mus81.Eme1霍利迪连接体解离酶是减数分裂交叉所必需的，但不是基因转换所必需的。
Genetics. 2003 Dec;165(4):2289-93. doi: 10.1093/genetics/165.4.2289.
2
Functional domains required for the Saccharomyces cerevisiae Mus81-Mms4 endonuclease complex formation and nuclear localization.酿酒酵母Mus81-Mms4核酸内切酶复合物形成及核定位所需的功能结构域。
DNA Repair (Amst). 2003 Dec 9;2(12):1435-47. doi: 10.1016/j.dnarep.2003.08.013.
3
Mus81 endonuclease localizes to nucleoli and to regions of DNA damage in human S-phase cells.Mus81核酸内切酶定位于人S期细胞的核仁及DNA损伤区域。
Mol Biol Cell. 2003 Dec;14(12):4826-34. doi: 10.1091/mbc.e03-05-0276.
4
Mek1 kinase activity functions downstream of RED1 in the regulation of meiotic double strand break repair in budding yeast.在芽殖酵母减数分裂双链断裂修复的调控中，Mek1激酶活性在RED1下游发挥作用。
Mol Biol Cell. 2004 Jan;15(1):11-23. doi: 10.1091/mbc.e03-07-0499. Epub 2003 Oct 31.
5
Holliday junctions in the eukaryotic nucleus: resolution in sight?真核细胞核中的霍利迪连接体：有望得到解决？
Trends Biochem Sci. 2003 Oct;28(10):548-57. doi: 10.1016/j.tibs.2003.08.011.
6
Generating crossovers by resolution of nicked Holliday junctions: a role for Mus81-Eme1 in meiosis.通过有切口的霍利迪连接体的拆分产生交叉：Mus81-Eme1在减数分裂中的作用
Mol Cell. 2003 Sep;12(3):761-74. doi: 10.1016/s1097-2765(03)00343-5.
7
The endogenous Mus81-Eme1 complex resolves Holliday junctions by a nick and counternick mechanism.内源性Mus81-Eme1复合物通过切口和反切口机制解析霍利迪连接体。
Mol Cell. 2003 Sep;12(3):747-59. doi: 10.1016/s1097-2765(03)00342-3.
8
Chromosome-wide control of meiotic crossing over in C. elegans.秀丽隐杆线虫减数分裂交叉的全染色体控制
Curr Biol. 2003 Sep 16;13(18):1641-7. doi: 10.1016/j.cub.2003.08.026.
9
The Mus81/Mms4 endonuclease acts independently of double-Holliday junction resolution to promote a distinct subset of crossovers during meiosis in budding yeast.Mus81/Mms4核酸内切酶在芽殖酵母减数分裂过程中独立于双Holliday连接体的解离发挥作用，以促进特定的交叉子集。
Genetics. 2003 May;164(1):81-94. doi: 10.1093/genetics/164.1.81.
10
The mechanism of Mus81-Mms4 cleavage site selection distinguishes it from the homologous endonuclease Rad1-Rad10.Mus81-Mms4切割位点选择机制使其有别于同源核酸内切酶Rad1-Rad10。
Mol Cell Biol. 2003 May;23(10):3487-96. doi: 10.1128/MCB.23.10.3487-3496.2003.
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