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Microbial etiology of periodontitis.

作者信息

Nishihara Tatsuji, Koseki Takeyoshi

机构信息

Department of Oral Microbiology, Kyushu Dental College, Fukuoka, Japan.

出版信息

Periodontol 2000. 2004;36:14-26. doi: 10.1111/j.1600-0757.2004.03671.x.

DOI:10.1111/j.1600-0757.2004.03671.x
PMID:15330940
Abstract
摘要

相似文献

1
Microbial etiology of periodontitis.牙周炎的微生物病因学
Periodontol 2000. 2004;36:14-26. doi: 10.1111/j.1600-0757.2004.03671.x.
2
The relationship between periodontal diseases and respiratory diseases.牙周疾病与呼吸道疾病之间的关系。
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Effects of nonsurgical periodontal therapy on the microbiota.非手术牙周治疗对微生物群的影响。
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Periodontitis as a biofilm infection.作为生物膜感染的牙周炎
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5
Oral ecology and its pathogenic relation with the origin of primary periodontitis associated with plaque.口腔生态学及其与菌斑相关性原发性牙周炎发病机制的关系。
Ann Biol Clin (Paris). 1991;49(6):338-44.
6
The etiology and pathogenesis of periodontitis.牙周炎的病因及发病机制。
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7
The microbial challenge in periodontitis.牙周炎中的微生物挑战。
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8
[Bacteria as a primary causative factor in inflammatory diseases of the periodontium].[细菌作为牙周炎炎症性疾病的主要致病因素]
Odontostomatol Proodos. 1979 Jan-Feb;33(1):9-30.
9
[The direct and indirect tissue-damaging action of periodontally pathogenic bacteria].[牙周病原菌的直接和间接组织损伤作用]
Minerva Stomatol. 1991 Apr;40(4):211-7.
10
Clinical relevance of the host responses of periodontitis.牙周炎宿主反应的临床相关性。
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Inducing cyclooxygenase-2 expression, prostaglandin E and prostaglandin F production of human dental pulp cells by activation of toll-like receptor-3, mitogen-activated protein kinase kinase/extracellular signal-regulated kinase and p38 signaling.通过激活Toll样受体3、丝裂原活化蛋白激酶激酶/细胞外信号调节激酶和p38信号通路，诱导人牙髓细胞中环氧合酶-2表达、前列腺素E和前列腺素F的产生。
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Periodontitis promotes bacterial extracellular vesicle-induced neuroinflammation in the brain and trigeminal ganglion.
牙周炎促进细菌细胞外囊泡诱导的大脑和三叉神经节神经炎症。
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Contemp Clin Dent. 2020 Jan-Mar;11(1):3-9. doi: 10.4103/ccd.ccd_448_19. Epub 2020 Jul 13.