Association of serum soluble heat shock protein 60 with toll-like receptor 4 polymorphism and severity of coronary artery disease.
作者信息
Mandal K, Afzal A R, Brecker S J D, Poloniecki J, Xu Q, Jahangiri M
出版信息
Heart. 2006 May;92(5):683-5. doi: 10.1136/hrt.2004.059170.
Abstract
摘要
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本文引用的文献
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Association of hypo-responsive toll-like receptor 4 variants with risk of myocardial infarction.低反应性Toll样受体4变体与心肌梗死风险的关联。
Eur Heart J. 2004 Aug;25(16):1447-53. doi: 10.1016/j.ehj.2004.05.004.
2
Autoimmune and inflammatory mechanisms in atherosclerosis.动脉粥样硬化中的自身免疫和炎症机制。
Annu Rev Immunol. 2004;22:361-403. doi: 10.1146/annurev.immunol.22.012703.104644.
3
Toll-like receptor-4 Asp299Gly polymorphism does not influence progression of atherosclerosis in patients with familial hypercholesterolaemia.Toll样受体4 Asp299Gly多态性不影响家族性高胆固醇血症患者动脉粥样硬化的进展。
Eur J Clin Invest. 2004 Feb;34(2):94-9. doi: 10.1111/j.1365-2362.2004.01303.x.
4
TLR4 Asp299Gly polymorphism is not associated with coronary artery stenosis.
Atherosclerosis. 2003 Sep;170(1):187-90. doi: 10.1016/s0021-9150(03)00286-7.
5
Monocytes heterozygous for the Asp299Gly and Thr399Ile mutations in the Toll-like receptor 4 gene show no deficit in lipopolysaccharide signalling.Toll样受体4基因中Asp299Gly和Thr399Ile突变的杂合单核细胞在脂多糖信号传导方面没有缺陷。
J Exp Med. 2003 Jun 16;197(12):1787-91. doi: 10.1084/jem.20022078. Epub 2003 Jun 9.
6
Toll-like receptor 4 polymorphisms and atherogenesis.Toll样受体4基因多态性与动脉粥样硬化的发生
N Engl J Med. 2002 Jul 18;347(3):185-92. doi: 10.1056/NEJMoa012673.
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The receptor for heat shock protein 60 on macrophages is saturable, specific, and distinct from receptors for other heat shock proteins.巨噬细胞上热休克蛋白60的受体是可饱和的、特异性的，且与其他热休克蛋白的受体不同。
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Serum soluble heat shock protein 60 is elevated in subjects with atherosclerosis in a general population.在普通人群中，动脉粥样硬化患者的血清可溶性热休克蛋白60水平升高。
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