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A STUDY OF MALIGNANT TUMOURS IN NIGERIA BY SHORT-TERM TISSUE CULTURE.
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Replication of viral deoxyribonucleic acid and breakdown of cellular deoxyribonucleic acid in Epstein-Barr virus infection.
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Preferential inhibition of herpes-group viruses by phosphonoacetic acid: effect on virus DNA synthesis and virus-induced DNA polymerase activity.
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Inhibition of productive replication of Epstein-Barr virus DNA by phosphonoacetic acid.
Virology. 1976 May;71(1):352-4. doi: 10.1016/0042-6822(76)90119-7.
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Differential effect of phosphonoacetic acid on the expression of Epstein-Barr viral antigens and virus production.
Proc Natl Acad Sci U S A. 1976 May;73(5):1745-8. doi: 10.1073/pnas.73.5.1745.
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Mutants of herpes simplex virus types 1 and 2 that are resistant to phosphonoacetic acid induce altered DNA polymerase activities in infected cells.
J Gen Virol. 1976 Apr;31(1):145-8. doi: 10.1099/0022-1317-31-1-145.
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Inhibition of Epstein-Barr virus DNA synthesis and late gene expression by phosphonoacetic acid.
J Virol. 1976 Apr;18(1):151-5. doi: 10.1128/JVI.18.1.151-155.1976.
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Synthesis of herpes simplex virus, vaccinia virus, and adenovirus DNA in isolated HeLa cell nuclei. I. Effect of viral-specific antisera and phosphonoacetic acid.
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Vertebrate DNA polymerases.
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Viral and cellular DNA synthesis in nuclei from human lymphocytes transformed by Epstein-Barr virus.
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