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本文引用的文献

1
In Vivo Effects of Clostridium perfringens Enteropathogenic Factors on the Rat Ileum.产气荚膜梭菌肠致病因子在大鼠回肠中的体内作用。
Infect Immun. 1974 Nov;10(5):1156-62. doi: 10.1128/iai.10.5.1156-1162.1974.
2
Substrate requirements for ion transport by rat intestine studied in vitro.体外研究大鼠肠道离子转运的底物需求。
Am J Physiol. 1960 Dec;199:1025-9. doi: 10.1152/ajplegacy.1960.199.6.1025.
3
Studies on the mechanism of oxidative phosphorylation. III. Phosphorylating particle types from beef heart.氧化磷酸化机制的研究。III. 牛心的磷酸化颗粒类型。
Biochim Biophys Acta. 1958 Jan;27(1):83-8. doi: 10.1016/0006-3002(58)90294-4.
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Determination of organic phosphorus compounds by phosphate analysis.通过磷酸盐分析测定有机磷化合物
Methods Biochem Anal. 1956;3:1-22. doi: 10.1002/9780470110195.ch1.
5
Purification and biochemical properties of Clostridium perfringens type A enterotoxin.A型产气荚膜梭菌肠毒素的纯化及生化特性
Infect Immun. 1972 Nov;6(5):662-73. doi: 10.1128/iai.6.5.662-673.1972.
6
Biological characteristics of Clostridium perfringens type A enterotoxin.A型产气荚膜梭菌肠毒素的生物学特性
Infect Immun. 1971 Aug;4(2):89-96. doi: 10.1128/iai.4.2.89-96.1971.
7
Biochemical effects of cholera enterotoxin: oxidative metabolism in the infant rabbit.霍乱肠毒素的生化效应：幼兔的氧化代谢
Am J Physiol. 1970 Mar;218(3):703-7. doi: 10.1152/ajplegacy.1970.218.3.703.
8
Effects of Clostridium perfringens enterotoxin on metabolic indexes of everted rat ileal sacs.产气荚膜梭菌肠毒素对大鼠外翻回肠囊代谢指标的影响
Infect Immun. 1975 Aug;12(2):274-80. doi: 10.1128/iai.12.2.274-280.1975.
9
Analysis of unidirectional fluxes of sodium during diarrhea induced by Clostridium perfringens enterotoxin in the rat terminal ileum.产气荚膜梭菌肠毒素诱导大鼠回肠末端腹泻期间钠单向通量的分析。
Infect Immun. 1975 Mar;11(3):526-9. doi: 10.1128/iai.11.3.526-529.1975.

产气荚膜梭菌肠毒素对线粒体呼吸的影响。

Effect of Clostridium perfrongens enterotoxin in mitochondrial respiration.

作者信息

McDonel J L, Duncan C L

出版信息

Infect Immun. 1977 Mar;15(3):999-1001. doi: 10.1128/iai.15.3.999-1001.1977.

DOI:10.1128/iai.15.3.999-1001.1977
PMID:192680
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC421473/
Abstract

Clostridium perfringens enterotoxin caused a 26 to 41% reduction in the rate of oxygen consumption by rat liver mitochondria when various tricarboxylic acid cycle intermediates were used as substrate. However, P/O ratios were unaltered.

摘要

当使用各种三羧酸循环中间产物作为底物时，产气荚膜梭菌肠毒素使大鼠肝线粒体的耗氧率降低了26%至41%。然而，磷氧比未发生改变。