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Ann Rheum Dis. 1990 Dec;49(12):973-5. doi: 10.1136/ard.49.12.973.
2
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Serum interleukin-6 levels and joint involvement in polyarticular and pauciarticular juvenile chronic arthritis.血清白细胞介素-6水平与多关节型及少关节型幼年慢性关节炎的关节受累情况
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2
Anti-endothelial cell antibodies are prevalent in juvenile idiopathic arthritis: implications for clinical disease course and pathogenesis.抗内皮细胞抗体在幼年特发性关节炎中普遍存在：对临床病程和发病机制的影响。
Rheumatol Int. 2007 May;27(7):655-60. doi: 10.1007/s00296-006-0276-3. Epub 2006 Dec 13.
3
Tissue plasminogen activator, plasminogen activator inhibitor-1 and von Willebrand factor in rheumatoid arthritis.类风湿关节炎中的组织型纤溶酶原激活剂、纤溶酶原激活剂抑制剂-1和血管性血友病因子
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Endothelial cell derived factors in relation to antibodies to SSA or SSB and to endothelial cells in Sjögren's syndrome.干燥综合征中与抗SSA或SSB抗体及内皮细胞相关的内皮细胞衍生因子
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本文引用的文献

1
Immunolocalization of von Willebrand protein in Weibel-Palade bodies of human endothelial cells.血管性血友病因子蛋白在人内皮细胞的魏尔-帕拉德小体中的免疫定位。
J Cell Biol. 1982 Oct;95(1):355-60. doi: 10.1083/jcb.95.1.355.
2
Consumption coagulopathy associated with systemic juvenile rheumatoid arthritis.与全身型幼年类风湿关节炎相关的消耗性凝血病。
J Pediatr. 1983 Dec;103(6):872-6. doi: 10.1016/s0022-3476(83)80704-5.
3
On the regulation and control of fibrinolysis. Edward Kowalski Memorial Lecture.论纤维蛋白溶解的调控。爱德华·科瓦尔斯基纪念讲座。
Thromb Haemost. 1980 Jun 18;43(2):77-89.
4
Acute hemorrhagic, hepatic, and neurologic manifestations in juvenile rheumatoid arthritis: possible relationship to drugs or infection.青少年类风湿关节炎的急性出血、肝脏及神经表现：与药物或感染的可能关系。
J Pediatr. 1985 Apr;106(4):561-6. doi: 10.1016/s0022-3476(85)80072-x.
5
Two different mechanisms in patients with venous thrombosis and defective fibrinolysis: low concentration of plasminogen activator or increased concentration of plasminogen activator inhibitor.静脉血栓形成且纤维蛋白溶解功能缺陷患者的两种不同机制：纤溶酶原激活物浓度低或纤溶酶原激活物抑制剂浓度升高。
Br Med J (Clin Res Ed). 1985 May 18;290(6480):1453-6. doi: 10.1136/bmj.290.6480.1453.
6
Low dose aspirin does not prevent fibrinolytic response to venous occlusion.低剂量阿司匹林不能预防对静脉闭塞的纤溶反应。
Biochem Pharmacol. 1986 Sep 15;35(18):3147-50. doi: 10.1016/0006-2952(86)90400-4.
7
Cachectin: more than a tumor necrosis factor.恶病质素：不仅仅是一种肿瘤坏死因子。
N Engl J Med. 1987 Feb 12;316(7):379-85. doi: 10.1056/NEJM198702123160705.
8
Deficient t-PA release and elevated PA inhibitor levels in patients with spontaneous or recurrent deep venous thrombosis.
Thromb Haemost. 1987 Feb 3;57(1):67-72.
9
Plasminogen activator inhibitors.纤溶酶原激活物抑制剂
Blood. 1987 Feb;69(2):381-7.
10
Enhanced interleukin 1 and depressed interleukin 2 production in juvenile arthritis.青少年关节炎中白细胞介素1升高和白细胞介素2产生降低。
J Rheumatol. 1986 Jun;13(3):598-603.

青少年慢性关节炎中纤维蛋白溶解活性降低。

Decreased fibrinolytic activity in juvenile chronic arthritis.

作者信息

Mussoni L, Pintucci G, Romano G, De Benedetti F, Massa M, Martini A

机构信息

Istituto di Ricerche, Farmacologiche Mario Negri, Milan, Italy.

出版信息

Ann Rheum Dis. 1990 Dec;49(12):973-5. doi: 10.1136/ard.49.12.973.

DOI:10.1136/ard.49.12.973
PMID:2125408
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1004289/
Abstract

The basal fibrinolytic activity in 17 children with active juvenile chronic arthritis (JCA) was investigated. It was found that patients with JCA, and particularly those with the systemic form, show decreased plasma fibrinolytic activity and a marked increase in plasminogen activator inhibitor. Additionally, it was found that patients with systemic JCA, but not those with the polyarticular or pauciarticular form, have increased circulating levels of tissue-type plasminogen activator, and endothelial cell protein, suggesting possible endothelial cell participation in systemic JCA.

摘要

对17名患有活动性青少年慢性关节炎（JCA）的儿童的基础纤溶活性进行了研究。发现JCA患者，尤其是全身型患者，血浆纤溶活性降低，纤溶酶原激活物抑制剂显著增加。此外，还发现全身型JCA患者，而非多关节型或少关节型患者，组织型纤溶酶原激活物和内皮细胞蛋白的循环水平升高，提示内皮细胞可能参与全身型JCA。