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吉兰-巴雷综合征严重自主神经功能障碍起病且恢复良好:一项临床及自主神经功能随访研究

Severe dysautonomic onset of Guillain-Barré syndrome with good recovery. A clinical and autonomic follow-up study.

作者信息

Cortelli P, Contin M, Lugaresi A, Baruzzi A, Montagna P

机构信息

Istituto di Clinica Neurologica, Università di Bologna.

出版信息

Ital J Neurol Sci. 1990 Apr;11(2):159-62. doi: 10.1007/BF02335559.

DOI:10.1007/BF02335559
PMID:2361852
Abstract

A 39 year old man with acute panautonomic and mild somatic neuropathy had severe postural hypotension 1 week after onset. Porphyric neuropathy was excluded. The final diagnosis was Guillain-Barré syndrome (GBS). After 2 months he began to recover progressively and after 9 months he presented asymptomatic postural hypotension. We consider the hypothesis of a spectrum of clinico-pathological entities at one end of which lies GBS with autonomic signs and at the other acute pure dysautonomia. The site of the autonomic lesion, might have been in post-ganglionic sympathetic fibers and vagus nerve.

摘要

一名39岁男性,患有急性全自主神经功能障碍和轻度躯体神经病变,发病1周后出现严重体位性低血压。排除了卟啉病性神经病变。最终诊断为吉兰-巴雷综合征(GBS)。2个月后他开始逐渐康复,9个月后出现无症状体位性低血压。我们考虑这样一种假说,即存在一系列临床病理实体,一端是伴有自主神经体征的GBS,另一端是急性单纯性自主神经功能障碍。自主神经病变的部位可能在节后交感神经纤维和迷走神经。

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Severe dysautonomic onset of Guillain-Barré syndrome with good recovery. A clinical and autonomic follow-up study.吉兰-巴雷综合征严重自主神经功能障碍起病且恢复良好:一项临床及自主神经功能随访研究
Ital J Neurol Sci. 1990 Apr;11(2):159-62. doi: 10.1007/BF02335559.
2
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本文引用的文献

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The mechanism of hypertension in the Guillain-Barré syndrome.吉兰-巴雷综合征中高血压的机制。
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