吞噬细胞呼吸爆发的激活与调节的分子基础。
Molecular basis of activation and regulation of the phagocyte respiratory burst.
作者信息
Hurst N P
出版信息
Ann Rheum Dis. 1987 Apr;46(4):265-72. doi: 10.1136/ard.46.4.265.
Abstract
The molecular basis of activation and regulation of the phagocyte respiratory burst is discussed with particular reference to the role of inositol phospholipid hydrolysis, guanine nucleotide coupling proteins, and activation of protein kinase C.
摘要
本文特别参考肌醇磷脂水解、鸟嘌呤核苷酸偶联蛋白和蛋白激酶C激活的作用，探讨了吞噬细胞呼吸爆发激活与调节的分子基础。
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本文引用的文献
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Oxidation-reduction properties of the cytochrome b found in the plasma-membrane fraction of human neutrophils. A possible oxidase in the respiratory burst.人中性粒细胞质膜组分中细胞色素b的氧化还原特性。呼吸爆发中一种可能的氧化酶。
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Biochemical signal transmitted by Fc gamma receptors: phospholipase A2 activity of Fc gamma 2b receptor of murine macrophage cell line P388D1.由Fcγ受体传递的生化信号：小鼠巨噬细胞系P388D1的Fcγ2b受体的磷脂酶A2活性
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Activation of the respiratory burst enzyme in human polymorphonuclear leukocytes by chemoattractants and other soluble stimuli. Evidence that the same oxidase is activated by different transductional mechanisms.趋化因子和其他可溶性刺激物对人多形核白细胞中呼吸爆发酶的激活作用。有证据表明，同一氧化酶可通过不同的转导机制被激活。
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