Bluming A Z, Cohen H G, Saxon A
Am J Med. 1979 Sep;67(3):421-8. doi: 10.1016/0002-9343(79)90788-5.
Two patients with angioimmunoblastic lymphadenopathy with dysproteinemia (AILD) were studied. Both patients had marked increases in all three major immunoglobulin classes, and both lacked suppressor cell activity in vitro. These findings are consistent with the theory that AILD is a defectively regulated immune response to an unidentified antigen(s) and could provide clues to the pathogenesis of other lymphoproliferative disorders as well.
对两名血管免疫母细胞性淋巴结病伴蛋白异常血症（AILD）患者进行了研究。两名患者的所有三种主要免疫球蛋白类别均显著增加，且两者在体外均缺乏抑制细胞活性。这些发现与AILD是对一种未明抗原的免疫反应调节缺陷的理论相符，并且也可能为其他淋巴增生性疾病的发病机制提供线索。
