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Plin5,糖尿病性心肌病的新靶点。

Plin5, a New Target in Diabetic Cardiomyopathy.

作者信息

Cui Xiangning, Wang Jingwu, Zhang Yang, Wei Jianliang, Wang Yan

机构信息

Department of Cardiovascular, Guang'anmen Hospital, China Academy of Chinese Medical Sciences, Beijing 100053, China.

Clinical Education Division, Shandong University of Traditional Chinese Medicine, Jinan, Shandong 250000, China.

出版信息

Oxid Med Cell Longev. 2022 Apr 25;2022:2122856. doi: 10.1155/2022/2122856. eCollection 2022.

Abstract

Abnormal lipid accumulation is commonly observed in diabetic cardiomyopathy (DC), which can create a lipotoxic microenvironment and damage cardiomyocytes. Lipid toxicity is an important pathogenic factor due to abnormal lipid accumulation in DC. As a lipid droplet (LD) decomposition barrier, Plin5 can protect LDs from lipase decomposition and regulate lipid metabolism, which is involved in the occurrence and development of cardiovascular diseases. In recent years, studies have shown that Plin5 expression is involved in the pathogenesis of DC lipid toxicity, such as oxidative stress, mitochondrial dysfunction, endoplasmic reticulum (ER) stress, and insulin resistance (IR) and has become a key target of DC research. Therefore, understanding the relationship between Plin5 and DC progression as well as the mechanism of this process is crucial for developing new therapeutic approaches and exploring new therapeutic targets. This review is aimed at exploring the latest findings and roles of Plin5 in lipid metabolism and DC-related pathogenesis, to explore possible clinical intervention approaches.

摘要

异常脂质蓄积在糖尿病性心肌病(DC)中普遍存在,这会产生脂毒性微环境并损害心肌细胞。由于DC中脂质异常蓄积,脂质毒性是一个重要的致病因素。作为一种脂滴(LD)分解屏障,Plin5可以保护脂滴免受脂肪酶分解并调节脂质代谢,其参与心血管疾病的发生和发展。近年来,研究表明Plin5表达参与DC脂质毒性的发病机制,如氧化应激、线粒体功能障碍、内质网(ER)应激和胰岛素抵抗(IR),并已成为DC研究的关键靶点。因此,了解Plin5与DC进展之间的关系以及这一过程的机制对于开发新的治疗方法和探索新的治疗靶点至关重要。本综述旨在探讨Plin5在脂质代谢和DC相关发病机制中的最新发现和作用,以探索可能的临床干预方法。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d45a/9060988/ca28c96f8c1d/OMCL2022-2122856.001.jpg

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