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J Neurol Neurosurg Psychiatry. 1973 Aug;36(4):604-6. doi: 10.1136/jnnp.36.4.604.
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本文引用的文献

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AN ENCEPHALITOGENIC PROTEIN OF HUMAN ORIGIN: SOME CHEMICAL AND BIOLOGICAL PROPERTIES.一种人类源致脑炎蛋白:一些化学和生物学特性
Ann N Y Acad Sci. 1965 Mar 31;122:182-98. doi: 10.1111/j.1749-6632.1965.tb20202.x.
2
THE AKR THYMIC ANTIGEN AND ITS DISTRIBUTION IN LEUKEMIAS AND NERVOUS TISSUES.AKR胸腺抗原及其在白血病和神经组织中的分布。
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DELAYED HYPERSENSITIVITY IN VITRO. I. THE SPECIFICITY OF INHIBITION OF CELL MIGRATION BY ANTIGENS.体外迟发型超敏反应。I. 抗原对细胞迁移抑制作用的特异性。
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Response of human blood lymphocytes to tuberculin PPD in tissue culture.人血淋巴细胞在组织培养中对结核菌素纯蛋白衍生物的反应。
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Mechanism of a reaction in vitro associated with delayed-type hypersensitivity.体外与迟发型超敏反应相关的反应机制。
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6
Mouse thymic iso-antigens.小鼠胸腺同种抗原。
Nature. 1966 Jan 29;209(5022):521-3. doi: 10.1038/209521b0.
7
Lymphocyte transformation in vitro measured by tritiated thymidine uptake. I. Lymphocyte culture techniques.通过氚标记胸腺嘧啶核苷摄取法体外测定淋巴细胞转化。I. 淋巴细胞培养技术。
Int Arch Allergy Appl Immunol. 1970;37(5):506-31. doi: 10.1159/000230240.
8
Lymphocyte-antigen interaction in electrophoretic mobility test for cellular sensitization.细胞致敏电泳迁移率试验中的淋巴细胞-抗原相互作用。
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9
The mechanism of antigen-induced electrophoretic mobility reduction of guinea-pig macrophages.豚鼠巨噬细胞抗原诱导的电泳迁移率降低的机制。
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10
Delayed hypersensitivity studies: some applications of cell electrophoresis.迟发型超敏反应研究:细胞电泳的一些应用
J R Coll Physicians Lond. 1972 Apr;6(3):316-26.

多发性硬化症及其他神经系统疾病中淋巴细胞对胸腺和淋巴结抗原的致敏作用。

Lymphocyte sensitization to thymus and lymph node antigen in multiple sclerosis and other neurological diseases.

作者信息

Field E J, Caspary E A

出版信息

J Neurol Neurosurg Psychiatry. 1973 Aug;36(4):604-6. doi: 10.1136/jnnp.36.4.604.

DOI:10.1136/jnnp.36.4.604
PMID:4125963
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC494416/
Abstract

Circulating lymphocytes from patients with multiple sclerosis are sensitized to saline homogenate of human thymus. They also show a minor degree of sensitization to lymph node extract. The sensitization to thymus is greater in multiple sclerosis than it is in other neurological disease (except dementia paralytica) and this may be related to the degree of parenchymatous destruction which provides the antigen stimulus or to astroglial overgrowth in these diseases. The observations support the view that human brain and thymus may share antigen(s) of the type known as ø-antigen in mice.

摘要

多发性硬化症患者的循环淋巴细胞对人胸腺盐水匀浆敏感。它们对淋巴结提取物也有轻微程度的敏感。多发性硬化症患者对胸腺的敏感性高于其他神经系统疾病(除麻痹性痴呆外),这可能与提供抗原刺激的实质破坏程度有关,或者与这些疾病中星形胶质细胞过度生长有关。这些观察结果支持这样一种观点,即人类大脑和胸腺可能共享在小鼠中被称为ø抗原的抗原类型。