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Activation of guanyl cyclase and intracellular cyclic GMP by fibroblast growth factor.

作者信息

Rudland P S, Gospodarowicz D, Seifert W

出版信息

Nature. 1974 Aug 30;250(5469):741-2, 773-4. doi: 10.1038/250741a0.

DOI:10.1038/250741a0
PMID:4153265
Abstract
摘要

相似文献

1
Activation of guanyl cyclase and intracellular cyclic GMP by fibroblast growth factor.
Nature. 1974 Aug 30;250(5469):741-2, 773-4. doi: 10.1038/250741a0.
2
Growth control in cultured mouse fibroblasts: induction of the pleiotypic and mitogenic responses by a purified growth factor.培养的小鼠成纤维细胞中的生长控制:一种纯化生长因子诱导的多型性和促有丝分裂反应。
Proc Natl Acad Sci U S A. 1974 Jul;71(7):2600-4. doi: 10.1073/pnas.71.7.2600.
3
[Disordered regulation of cell membrane adenyl cyclase and guanyl cyclase activity as a possible factor in carcinogenesis].[细胞膜腺苷酸环化酶和鸟苷酸环化酶活性调节紊乱作为致癌作用的一个可能因素]
Vopr Onkol. 1978;24(10):28-31.
4
Pharmacological control of the synthesis and metabolism of cyclic nucleotides.环核苷酸合成与代谢的药理学控制
Adv Pharmacol Chemother. 1977;14:189-283. doi: 10.1016/s1054-3589(08)60189-1.
5
Cyclic GMP and cyclic AMP levels in normal and transformed fibroblasts.正常和成纤维细胞中环状鸟苷单磷酸和环状腺苷单磷酸水平
Nature. 1974 Oct 4;251(5474):417-9. doi: 10.1038/251417a0.
6
Properties and regulation of guanylate cyclase and some proposed functions for cyclic GMP.
Adv Cyclic Nucleotide Res. 1979;11:175-204.
7
Possible involvement of cyclic GMP in growth control of cultured mouse cells.环磷酸鸟苷可能参与培养的小鼠细胞的生长控制。
Nature. 1974 Mar 8;248(5444):138-40. doi: 10.1038/248138a0.
8
Properties and oxidative regulation of guanylate cyclase.
J Cyclic Nucleotide Res. 1977 Dec;3(6):381-91.
9
Atrial natriuretic factor and sodium nitroprusside increase cyclic GMP in cultured rat lung fibroblasts by activating different forms of guanylate cyclase.心房利钠因子和硝普钠通过激活不同形式的鸟苷酸环化酶增加培养的大鼠肺成纤维细胞中的环磷酸鸟苷。
Biochem J. 1987 May 15;244(1):69-74. doi: 10.1042/bj2440069.
10
[Changed equilibrium between the sympathetic-adenyl cyclase-cyclic AMP-PGE system and the cholinergic-guanyl cyclase-cyclic GMP-PGF2 alpha system as a pathogenetic mechanism of bronchial asthma].[交感神经-腺苷酸环化酶-环磷酸腺苷-前列腺素E系统与胆碱能-鸟苷酸环化酶-环磷酸鸟苷-前列腺素F2α系统之间平衡的改变作为支气管哮喘的发病机制]
G Clin Med. 1980 Jun;61(6):444-8.

引用本文的文献

1
Biotin analogs activate guanylate cyclase.生物素类似物可激活鸟苷酸环化酶。
Mol Cell Biochem. 1984;60(2):109-14. doi: 10.1007/BF00222480.
2
Guanylate cyclase activity in Escherichia coli mutants defective in adenylate cyclase.腺苷酸环化酶缺陷的大肠杆菌突变体中的鸟苷酸环化酶活性。
J Bacteriol. 1981 Sep;147(3):931-4. doi: 10.1128/jb.147.3.931-934.1981.
3
Cyclic nucleotides and growth control in cultured mouse cells: correlation of changes in intracellular 3':5' cGMP concentration with a specific phase of the cell cycle.培养的小鼠细胞中的环核苷酸与生长控制:细胞内3':5'-环鸟苷酸浓度变化与细胞周期特定阶段的相关性。
Proc Natl Acad Sci U S A. 1974 Dec;71(12):4920-4. doi: 10.1073/pnas.71.12.4920.
4
An experimental trial of cyclic nucleotides on multicellular spheroids derived from human brain tumours.
J Neurooncol. 1987;5(1):83-9. doi: 10.1007/BF00162770.
5
Stimulation of guanylate cyclase and RNA polymerase II activities in HeLa cells and fibroblasts by biotin.生物素对HeLa细胞和成纤维细胞中鸟苷酸环化酶及RNA聚合酶II活性的刺激作用。
Mol Cell Biochem. 1988 Jan;79(1):47-55. doi: 10.1007/BF00229397.
6
The different roles of serum and calcium in the control of proliferation of BALB/c 3T3 mouse cells.血清和钙在控制BALB/c 3T3小鼠细胞增殖中的不同作用。
In Vitro. 1976 Feb;12(2):120-3. doi: 10.1007/BF02796358.
7
Thymidine kinase, DNA synthesis and cancer.胸苷激酶、DNA合成与癌症。
Mol Cell Biochem. 1976 Jun 15;11(3):161-82. doi: 10.1007/BF01744997.
8
Influence of prostaglandins on connective tissue cell growth and function.前列腺素对结缔组织细胞生长和功能的影响。
Naunyn Schmiedebergs Arch Pharmacol. 1977;297 Suppl 1:S89-93. doi: 10.1007/BF00587790.
9
Increased particulate and decreased soluble guanylate cyclase activity in regenerating liver, fetal liver, and hepatoma.再生肝、胎儿肝和肝癌中颗粒增多及可溶性鸟苷酸环化酶活性降低。
Proc Natl Acad Sci U S A. 1975 May;72(5):1965-9. doi: 10.1073/pnas.72.5.1965.
10
Response of lymphocyte guanyl cyclase to propranolol, noradrenaline, thymoxamine, and acetylcholine in extrinsic bronchial asthma.外源性支气管哮喘中淋巴细胞鸟苷酸环化酶对普萘洛尔、去甲肾上腺素、胸腺氧胺和乙酰胆碱的反应
Br Med J. 1975 May 17;2(5967):357-9. doi: 10.1136/bmj.2.5967.357.