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Gut. 1984 Nov;25(11):1230-3. doi: 10.1136/gut.25.11.1230.
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Effect of truncal vagotomy on functional and morphological changes produced by duodenogastric reflux.迷走神经干切断术对十二指肠-胃反流所致功能和形态学改变的影响。
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本文引用的文献

1
Functional changes in acid secretion produced by duodenogastric reflux.十二指肠-胃反流引起的胃酸分泌功能变化。
Gut. 1980 May;21(5):413-7. doi: 10.1136/gut.21.5.413.
2
Gastric morphological and functional changes produced by bile in the canine stomach.胆汁对犬胃造成的胃形态学和功能变化。
Eur Surg Res. 1981;13(2):125-33. doi: 10.1159/000128178.
3
The effect of duodeno-gastric reflux on serum gastrin levels in the dog.十二指肠-胃反流对犬血清胃泌素水平的影响。
Eur Surg Res. 1980;12(6):403-7. doi: 10.1159/000128147.
4
Bile acid and lysolecithin concentrations in the stomach in patients with duodenal ulcer before operation and after treatment by highly selective vagotomy, partial gastrectomy, or truncal vagotomy and drainage.十二指肠溃疡患者在手术前以及经高选择性迷走神经切断术、胃部分切除术或迷走神经干切断术加引流术治疗后的胃内胆汁酸和溶血卵磷脂浓度。
Gut. 1982 Jul;23(7):569-77. doi: 10.1136/gut.23.7.569.
5
Effect of truncal vagotomy on functional and morphological changes produced by duodenogastric reflux.迷走神经干切断术对十二指肠-胃反流所致功能和形态学改变的影响。
Dig Dis Sci. 1982 Jul;27(7):609-14. doi: 10.1007/BF01297217.
6
Antral gastrin-producing G-cells and somatostatin-producing D-cells in different states of gastric acid secretion.处于不同胃酸分泌状态下的胃窦部产胃泌素G细胞和产生长抑素D细胞。
Gut. 1982 Apr;23(4):285-91. doi: 10.1136/gut.23.4.285.
7
The kinetics of somatostatin inhibition of gastric acid secretion.生长抑素抑制胃酸分泌的动力学。
Regul Pept. 1980 Dec;1(3):245-51. doi: 10.1016/0167-0115(80)90274-8.
8
Inhibitory effect of somatostatin on gastric acid secretion and serum gastrin in dogs with and without duodenogastric reflux.生长抑素对伴有和不伴有十二指肠-胃反流犬胃酸分泌及血清胃泌素的抑制作用
Gut. 1980 Nov;21(11):996-1001. doi: 10.1136/gut.21.11.996.
9
Duodeno-gastric reflux--a common factor in pathogenesis of gastric and duodenal ulcer.十二指肠-胃反流——胃及十二指肠溃疡发病机制中的一个常见因素。
Lancet. 1980 Nov 29;2(8205):1166-8. doi: 10.1016/s0140-6736(80)92598-2.
10
Increased reflux of bile into the stomach in patients with gastric ulcer.胃溃疡患者胆汁反流至胃内的情况增加。
Gastroenterology. 1969 Sep;57(3):241-52.

犬十二指肠-胃反流对生长抑素释放的抑制作用。

Suppression of somatostatin release by duodenogastric reflux in dogs.

作者信息

Thomas W E, Ardill J, Buchanan K D

出版信息

Gut. 1984 Nov;25(11):1230-3. doi: 10.1136/gut.25.11.1230.

DOI:10.1136/gut.25.11.1230
PMID:6149979
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1432327/
Abstract

The effect of duodenogastric reflux on systemic and portal venous blood concentrations of somatostatin has been studied in the dog. Duodenogastric reflux suppressed somatostatin concentrations in both systemic and portal venous blood, but this did not occur when bile alone was diverted into the stomach. The suppression was also much less marked when truncal vagotomy accompanied the reflux. These findings suggest that altered somatostatin activity may play a part in the production of the pathophysiological changes occurring in clinical conditions such as peptic ulceration, in which there is an increase in duodenogastric reflux.

摘要

十二指肠-胃反流对犬体循环和门静脉血中生长抑素浓度的影响已得到研究。十二指肠-胃反流可抑制体循环和门静脉血中生长抑素的浓度，但单纯将胆汁引流至胃内时则不会出现这种情况。当迷走神经干切断术与反流同时存在时，这种抑制作用也明显减弱。这些发现提示，生长抑素活性的改变可能在诸如消化性溃疡等临床疾病所发生的病理生理变化中起作用，在这些疾病中十二指肠-胃反流增加。