Centre National de la Recherche Scientifique, UMR 7357 ICube CNRS and Université de Strasbourg Hôpitaux Universitaires Faculté de Médecine, Pavillon Clovis Vincent, 4, rue Kirschleger, 67085 Strasbourg Cedex, France.
Int J Environ Res Public Health. 2020 May 6;17(9):3240. doi: 10.3390/ijerph17093240.
Environmental studies, metabolic research, and state of the art research in neurobiology point towards the reduced amount of natural day and sunlight exposure of the developing child, as a consequence of increasingly long hours spent indoors online, as the single unifying source of a whole set of health risks identified worldwide, as is made clear in this review of currently available literature. Over exposure to digital environments, from abuse to addiction, now concerns even the youngest (ages 0 to 2) and triggers, as argued on the basis of clear examples herein, a chain of interdependent negative and potentially long-term metabolic changes. This leads to a deregulation of the serotonin and dopamine neurotransmitter pathways in the developing brain, currently associated with online activity abuse and/or internet addiction, and akin to that found in severe substance abuse syndromes. A general functional working model is proposed under the light of evidence brought to the forefront in this review.
环境研究、代谢研究和神经生物学的最新研究都指向了一个问题,那就是由于儿童在室内上网的时间越来越长,他们接触自然光和日照的时间减少了。这是造成全世界一系列健康风险的单一的、统一的根源,这在对现有文献的综述中已经清楚地表明了。过度暴露于数字环境,无论是滥用还是成瘾,现在甚至连最小的(0 到 2 岁)儿童也受到了影响,并且根据本文中的明确例子,可以触发一系列相互依存的负面的、潜在的长期代谢变化。这会导致发育中的大脑中血清素和多巴胺神经递质通路的失调,目前与在线活动滥用和/或网络成瘾有关,类似于在严重的物质滥用综合征中发现的情况。在本文综述提出的证据的基础上,提出了一个通用的功能工作模型。