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本文引用的文献

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THE PREPARATION OF I-131-LABELLED HUMAN GROWTH HORMONE OF HIGH SPECIFIC RADIOACTIVITY.高比放射性碘-131标记人生长激素的制备
Biochem J. 1963 Oct;89(1):114-23. doi: 10.1042/bj0890114.
2
Chemotaxis in yersinia arthritis. HLA-B27 positive neutrophils show high stimulated motility in vitro.耶尔森菌关节炎中的趋化作用。HLA - B27阳性中性粒细胞在体外表现出高刺激运动性。
Arthritis Rheum. 1980 Sep;23(9):1036-44. doi: 10.1002/art.1780230911.
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The in-vivo metabolism of C3 in ankylosing spondylitis.强直性脊柱炎中C3的体内代谢
Ann Rheum Dis. 1982 Feb;41(1):86-9. doi: 10.1136/ard.41.1.86.
4
Chemotaxis in yersinia arthritis. In vitro stimulation of neutrophil migration by HLA--B27 positive and negative sera.耶尔森菌关节炎中的趋化作用。HLA - B27阳性和阴性血清对中性粒细胞迁移的体外刺激。
Arthritis Rheum. 1982 Jun;25(6):655-61. doi: 10.1002/art.1780250608.
5
Studies on the purification and structure-functional relationships of murine lymphocyte activating factor (Interleukin 1).小鼠淋巴细胞激活因子（白细胞介素1）的纯化及结构-功能关系研究
Mol Immunol. 1980 May;17(5):571-7. doi: 10.1016/0161-5890(80)90155-8.
6
Why HLA-B27: an analysis based on two animal models.为何是HLA - B27：基于两种动物模型的分析
Ann Intern Med. 1981 Feb;94(2):261-3. doi: 10.7326/0003-4819-94-2-261.
7
Polymorphonuclear cell motility, ankylosing spondylitis, and HLA B27.多形核细胞运动、强直性脊柱炎与HLA B27
Ann Rheum Dis. 1984 Apr;43(2):279-84. doi: 10.1136/ard.43.2.279.
8
Enhanced neutrophil migration in vivo HLA B27 positive subjects.HLA B27阳性受试者体内中性粒细胞迁移增强。
Ann Rheum Dis. 1984 Apr;43(2):181-5. doi: 10.1136/ard.43.2.181.
9
Separation of blood leucocytes, granulocytes and lymphocytes.血液白细胞、粒细胞和淋巴细胞的分离。
Tissue Antigens. 1974;4(4):269-74.
10
Enhanced prostanoid release from monocytes of patients with rheumatoid arthritis and active systemic lupus erythematosus.类风湿关节炎和活动性系统性红斑狼疮患者单核细胞中前列腺素释放增加。
Ann Rheum Dis. 1985 Jul;44(7):438-45. doi: 10.1136/ard.44.7.438.

既往有耶尔森菌关节炎患者单核细胞产生肿瘤坏死因子和白细胞介素1的情况。

Production of tumour necrosis factor and interleukin 1 by monocytes of patients with previous Yersinia arthritis.

作者信息

Repo H, Jäättelä M, Leirisalo-Repo M, Hurme M

机构信息

Department of Bacteriology and Immunology, University of Helsinki, Finland.

出版信息

Clin Exp Immunol. 1988 Jun;72(3):410-4.

PMID:3262459
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1541576/
Abstract

We studied production of tumour necrosis factor (TNF) and interleukin 1 (IL-1) by using purified peripheral blood monocytes of patients with previous yersinia arthritis (YA) and of healthy HLA-B27 positive and negative controls. Lipopolysaccharide-exposed cells of HLA-B27 positive and negative patients, and those of HLA-B27 positive controls, generated significantly more TNF than did HLA-B27 negative control cells. There was a positive correlation between the levels of TNF and IL-1. Our results give credence to the view that augmented production of phlogistic mediators may contribute to inflammatory symptoms in patients with HLA-B27 associated disease.

摘要

我们采用纯化的外周血单核细胞，对曾患耶尔森菌关节炎（YA）的患者以及健康的HLA - B27阳性和阴性对照者进行研究，以检测肿瘤坏死因子（TNF）和白细胞介素1（IL - 1）的产生情况。HLA - B27阳性和阴性患者以及HLA - B27阳性对照者的脂多糖刺激细胞所产生的TNF，显著多于HLA - B27阴性对照细胞。TNF和IL - 1水平之间存在正相关。我们的结果支持这样一种观点，即炎症介质产生增加可能导致HLA - B27相关疾病患者出现炎症症状。