Liu Chang, Rotaru Kristian, Lee Rico S C, Tiego Jeggan, Suo Chao, Yücel Murat, Albertella Lucy
1BrainPark, Turner Institute for Brain and Mental Health and School of Psychological Sciences, Monash University, Australia.
2Monash Business School, Monash University, Australia.
J Behav Addict. 2021 Apr 27;10(3):534-9. doi: 10.1556/2006.2021.00027.
Researchers are only just beginning to understand the neurocognitive drivers of addiction-like eating behaviours, a highly distressing and relatively common condition. Two constructs have been consistently linked to addiction-like eating: distress-driven impulsivity and cognitive inflexibility. Despite a large body of addiction research showing that impulsivity-related traits can interact with other risk markers to result in an especially heightened risk for addictive behaviours, no study to date has examined how distress-driven impulsivity interacts with cognitive inflexibility in relation to addiction-like eating behaviours. The current study examines the interactive contribution of distress-driven impulsivity and cognitive inflexibility to addiction-like eating behaviours.
One hundred and thirty-one participants [mean age 21 years (SD = 2.3), 61.8% female] completed the modified Yale Food Addiction Scale, the S-UPPS-P impulsivity scale, and a cognitive flexibility task. A bootstrap method was used to examine the associations between distress-driven impulsivity, cognitive inflexibility, and their interaction with addiction-like eating behaviours.
There was a significant interaction effect between distress-driven impulsivity and cognitive flexibility (P = 0.03). The follow-up test revealed that higher distress-driven impulsivity was associated with more addiction-like eating behaviours among participants classified as cognitively inflexible only.
The current findings shed light on the mechanisms underlying addiction-like eating behaviours, including how traits and cognition might interact to drive them. The findings also suggest that interventions that directly address distress-driven impulsivity and cognitive inflexibility might be effective in reducing risk for addiction-like eating and related disorders.
研究人员才刚刚开始了解成瘾性饮食行为的神经认知驱动因素，这是一种极为困扰且相对常见的状况。有两种结构一直与成瘾性饮食相关联：痛苦驱动的冲动性和认知灵活性不足。尽管大量成瘾研究表明，与冲动性相关的特质可与其他风险标志物相互作用，从而导致成瘾行为的风险尤其升高，但迄今为止，尚无研究探讨痛苦驱动的冲动性与认知灵活性不足在成瘾性饮食行为方面是如何相互作用的。本研究考察了痛苦驱动的冲动性和认知灵活性不足对成瘾性饮食行为的交互作用。
131名参与者[平均年龄21岁（标准差=2.3），61.8%为女性]完成了改良版耶鲁食物成瘾量表、S-UPPS-P冲动性量表以及一项认知灵活性任务。采用自助法来考察痛苦驱动的冲动性、认知灵活性不足及其与成瘾性饮食行为之间的关联。
痛苦驱动的冲动性与认知灵活性之间存在显著的交互作用（P=0.03）。后续测试显示，仅在被归类为认知灵活性不足的参与者中，较高的痛苦驱动冲动性与更多的成瘾性饮食行为相关。
当前研究结果揭示了成瘾性饮食行为背后的机制，包括特质和认知可能如何相互作用以驱动这些行为。研究结果还表明，直接针对痛苦驱动的冲动性和认知灵活性不足的干预措施可能有效降低成瘾性饮食及相关障碍的风险。
