• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

1985 年至 2020 年糖尿病肾病肾纤维化的文献计量学分析。

Bibliometric Analysis of Renal Fibrosis in Diabetic Kidney Disease From 1985 to 2020.

机构信息

Endocrinology Department, Guang'anmen Hospital, China Academy of Chinese Medical Sciences, Beijing, China.

Endocrinology Department, Guang'anmen Hospital, Beijing University of Chinese Medicine, Beijing, China.

出版信息

Front Public Health. 2022 Feb 4;10:767591. doi: 10.3389/fpubh.2022.767591. eCollection 2022.

DOI:10.3389/fpubh.2022.767591
PMID:35186833
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8855938/
Abstract

BACKGROUND

Diabetic renal fibrosis (DRF) is an irreversible renal pathological change in the end-stage of diabetic kidney disease (DKD), which plays a significant role in the development and deterioration of the disease. However, data for bibliometric analysis of renal fibrosis in DKD is currently missing. This study aimed to provide a comprehensive and visualized view of DRF research and lay the foundation for further studies.

MATERIALS AND METHODS

Firstly, the data was collected from the Web of Science Core Collection (WoSCC) database. Secondly, the Web of Science analytic tool was performed to analyze publication years, authors, countries/regions, organizations, and citation frequency. Finally, CiteSpace was employed to construct a visualization bibliometric network to reveal the emerging trends and hotspots of DRF.

RESULTS

A total of 3,821 publications from 1985 to 2020 were included in this study. The number of publications has maintained a growth trend since 2003. Cooper is the most prolific author in this field, and the ranking as first place compared with other journals. In terms of the number of publications, China contributed the most to DRF. Monash University is the organization that published the most papers. The top 5 clusters of keyword co-appearance are "chronic kidney disease", "primary biliary cirrhosis", "receptor", "TGF-beta", "renal tubulointerstitium". The top 5 clusters of reference co-citation are "microRNAs", "bone morphogenetic protein", "hypertrophy", "glomerulosclerosis", "diabetic kidney disease". The strongest citation burst of keyword is "diabetic kidney disease" and the strongest burst of cited reference is "Meng, 2016".

CONCLUSIONS

The present study analyzed the research hotspots, Frontiers, and development trend of DRF and have important implications for future research.

摘要

背景

糖尿病肾病（DKD）终末期的糖尿病肾纤维化（DRF）是一种不可逆转的肾脏病理改变，在疾病的发生和恶化中起着重要作用。然而，目前缺乏对 DKD 肾纤维化的文献计量学分析数据。本研究旨在为 DRF 研究提供一个全面和可视化的视图，并为进一步的研究奠定基础。

材料与方法

首先，从 Web of Science 核心合集（WoSCC）数据库中收集数据。其次，使用 Web of Science 分析工具分析出版物年份、作者、国家/地区、组织和引文频率。最后，使用 CiteSpace 构建可视化文献计量网络，以揭示 DRF 的新兴趋势和热点。

结果

共纳入 1985 年至 2020 年的 3821 篇出版物。自 2003 年以来，出版物数量一直保持增长趋势。Cooper 是该领域最有影响力的作者，排名第一，优于其他期刊。就出版物数量而言，中国对 DRF 的贡献最大。Monash 大学是发表论文最多的机构。关键词共现的前 5 个聚类是“慢性肾脏病”、“原发性胆汁性肝硬化”、“受体”、“TGF-β”、“肾小管间质”。被引文献共被引的前 5 个聚类是“microRNAs”、“骨形态发生蛋白”、“肥大”、“肾小球硬化”、“糖尿病肾病”。关键词最强引文突现是“糖尿病肾病”，被引文献最强突现是“Meng, 2016”。

结论

本研究分析了 DRF 的研究热点、前沿和发展趋势，对未来的研究具有重要意义。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9a79/8855938/163fe91df437/fpubh-10-767591-g0009.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9a79/8855938/18b190032778/fpubh-10-767591-g0001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9a79/8855938/7f6853ac53ec/fpubh-10-767591-g0002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9a79/8855938/2b2e0e670bda/fpubh-10-767591-g0003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9a79/8855938/d4ba88b0e52d/fpubh-10-767591-g0004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9a79/8855938/9dd14611f8f3/fpubh-10-767591-g0005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9a79/8855938/50e564d24af2/fpubh-10-767591-g0006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9a79/8855938/ec5e8b51c30d/fpubh-10-767591-g0007.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9a79/8855938/4c98811b5c74/fpubh-10-767591-g0008.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9a79/8855938/163fe91df437/fpubh-10-767591-g0009.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9a79/8855938/18b190032778/fpubh-10-767591-g0001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9a79/8855938/7f6853ac53ec/fpubh-10-767591-g0002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9a79/8855938/2b2e0e670bda/fpubh-10-767591-g0003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9a79/8855938/d4ba88b0e52d/fpubh-10-767591-g0004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9a79/8855938/9dd14611f8f3/fpubh-10-767591-g0005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9a79/8855938/50e564d24af2/fpubh-10-767591-g0006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9a79/8855938/ec5e8b51c30d/fpubh-10-767591-g0007.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9a79/8855938/4c98811b5c74/fpubh-10-767591-g0008.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9a79/8855938/163fe91df437/fpubh-10-767591-g0009.jpg

相似文献

1
Bibliometric Analysis of Renal Fibrosis in Diabetic Kidney Disease From 1985 to 2020.1985 年至 2020 年糖尿病肾病肾纤维化的文献计量学分析。
Front Public Health. 2022 Feb 4;10:767591. doi: 10.3389/fpubh.2022.767591. eCollection 2022.
2
Evolution and global research trends of immunity in diabetic nephropathy: a bibliometric and visual analysis from 2004 to 2023.免疫在糖尿病肾病中的作用的演变和全球研究趋势：2004 年至 2023 年的文献计量和可视化分析。
Int Urol Nephrol. 2024 Oct;56(10):3307-3321. doi: 10.1007/s11255-024-04081-x. Epub 2024 May 17.
3
The 100 top-cited articles in diabetic kidney disease: a bibliometric analysis.100 篇高被引糖尿病肾病文献的计量分析。
Ren Fail. 2021 Dec;43(1):781-795. doi: 10.1080/0886022X.2021.1919528.
4
Tiny clue reveals the general trend: a bibliometric and visualized analysis of renal microcirculation.微小线索揭示总体趋势：肾微循环的文献计量学和可视化分析。
Ren Fail. 2024 Dec;46(1):2329249. doi: 10.1080/0886022X.2024.2329249. Epub 2024 Mar 14.
5
Global diabetic kidney disease research from 2000 to 2017: A bibliometric analysis.2000年至2017年全球糖尿病肾病研究：一项文献计量分析。
Medicine (Baltimore). 2019 Feb;98(6):e14394. doi: 10.1097/MD.0000000000014394.
6
The intellectual base and global trends in inflammation of diabetic kidney disease: a bibliometric analysis.糖尿病肾病炎症的知识基础和全球趋势：文献计量分析。
Ren Fail. 2023;45(2):2270061. doi: 10.1080/0886022X.2023.2270061. Epub 2023 Oct 23.
7
Bibliometric analysis of T-cells immunity in pulmonary hypertension from 1992 to 2022.1992 年至 2022 年肺动脉高压中 T 细胞免疫的文献计量学分析。
Immun Inflamm Dis. 2024 Jul;12(7):e1280. doi: 10.1002/iid3.1280.
8
Global trends and focuses of GLP-1RA in renal disease: a bibliometric analysis and visualization from 2005 to 2022.2005年至2022年GLP-1受体激动剂在肾脏疾病中的全球趋势与研究重点：一项文献计量分析与可视化研究
Naunyn Schmiedebergs Arch Pharmacol. 2023 Dec;396(12):3347-3361. doi: 10.1007/s00210-023-02575-6. Epub 2023 Jun 30.
9
Frontiers and hotspots evolution in anti-inflammatory studies for coronary heart disease: A bibliometric analysis of 1990-2022.冠心病抗炎研究的前沿与热点演变：1990 - 2022年文献计量分析
Front Cardiovasc Med. 2023 Feb 16;10:1038738. doi: 10.3389/fcvm.2023.1038738. eCollection 2023.
10
Global trends and hotspots of treatment for nonalcoholic fatty liver disease: A bibliometric and visualization analysis (2010-2023).全球非酒精性脂肪性肝病治疗的趋势和热点：文献计量学和可视化分析（2010-2023）。
World J Gastroenterol. 2023 Oct 7;29(37):5339-5360. doi: 10.3748/wjg.v29.i37.5339.

引用本文的文献

1
Cutting-edge and topical issues in the treatment of breast cancer with traditional Chinese medicine based on CiteSpace bibliometric analysis.基于CiteSpace文献计量分析的中医治疗乳腺癌前沿与热点问题
Medicine (Baltimore). 2024 Dec 6;103(49):e40784. doi: 10.1097/MD.0000000000040784.
2
Focus on podocytes: diabetic kidney disease and renal fibrosis - a global bibliometric analysis (2000-2024).聚焦足细胞：糖尿病肾病与肾纤维化——一项全球文献计量分析（2000 - 2024年）
Front Pharmacol. 2024 Nov 15;15:1454586. doi: 10.3389/fphar.2024.1454586. eCollection 2024.
3
Global research trends of MicroRNAs in rheumatoid arthritis: bibliometrics and visualization analysis.

本文引用的文献

1
Potential Targeting of Renal Fibrosis in Diabetic Kidney Disease Using MicroRNAs.利用微小RNA靶向治疗糖尿病肾病中的肾纤维化
Front Pharmacol. 2020 Nov 13;11:587689. doi: 10.3389/fphar.2020.587689. eCollection 2020.
2
Profibrotic Circulating Proteins and Risk of Early Progressive Renal Decline in Patients With Type 2 Diabetes With and Without Albuminuria.有和无白蛋白尿的2型糖尿病患者中促纤维化循环蛋白与早期进行性肾衰退风险
Diabetes Care. 2020 Nov;43(11):2760-2767. doi: 10.2337/dc20-0630. Epub 2020 Sep 4.
3
The Prediction of Infectious Diseases: A Bibliometric Analysis.
类风湿关节炎中微小RNA的全球研究趋势：文献计量学与可视化分析
Clin Rheumatol. 2025 Jan;44(1):53-66. doi: 10.1007/s10067-024-07250-6. Epub 2024 Nov 28.
4
Mapping knowledge structure and themes trends of non-surgical treatment in intervertebral disc degeneration.绘制椎间盘退变非手术治疗的知识结构和主题趋势。
Heliyon. 2024 Aug 28;10(17):e36509. doi: 10.1016/j.heliyon.2024.e36509. eCollection 2024 Sep 15.
5
Simultaneous detection of multiple urinary biomarkers in patients with early-stage diabetic kidney disease using Luminex liquid suspension chip technology.采用 Luminex 液相悬浮芯片技术同时检测早期糖尿病肾病患者的多种尿生物标志物。
Front Endocrinol (Lausanne). 2024 Aug 20;15:1443573. doi: 10.3389/fendo.2024.1443573. eCollection 2024.
6
The 100 top-cited articles on chronic kidney disease-mineral and bone disorder: A bibliometric analysis.100 篇高被引慢性肾脏病-矿物质和骨异常文献的计量学分析。
Medicine (Baltimore). 2024 Apr 19;103(16):e37835. doi: 10.1097/MD.0000000000037835.
7
Loss of Farnesoid X receptor (FXR) accelerates dysregulated glucose and renal injury in db/db mice.法尼醇 X 受体 (FXR) 的缺失会加速 db/db 小鼠中葡萄糖代谢紊乱和肾脏损伤。
PeerJ. 2023 Sep 29;11:e16155. doi: 10.7717/peerj.16155. eCollection 2023.
8
Long Non-coding RNA ZFAS1 Regulates Fibrosis and Scortosis in the Cell Model of Diabetic Nephropathy Through miR-525-5p/SGK1 Axis.长链非编码 RNA ZFAS1 通过 miR-525-5p/SGK1 轴调控糖尿病肾病细胞模型中的纤维化和硬化。
Appl Biochem Biotechnol. 2024 Jul;196(7):3731-3746. doi: 10.1007/s12010-023-04721-5. Epub 2023 Sep 28.
9
Research progress and hotspots on macrophages in osteoarthritis: A bibliometric analysis from 2009 to 2022.骨关节炎中巨噬细胞的研究进展和热点：2009 年至 2022 年的文献计量学分析。
Medicine (Baltimore). 2023 Aug 25;102(34):e34642. doi: 10.1097/MD.0000000000034642.
10
Global trends and focuses of GLP-1RA in renal disease: a bibliometric analysis and visualization from 2005 to 2022.2005年至2022年GLP-1受体激动剂在肾脏疾病中的全球趋势与研究重点：一项文献计量分析与可视化研究
Naunyn Schmiedebergs Arch Pharmacol. 2023 Dec;396(12):3347-3361. doi: 10.1007/s00210-023-02575-6. Epub 2023 Jun 30.
传染病预测：文献计量分析。
Int J Environ Res Public Health. 2020 Aug 27;17(17):6218. doi: 10.3390/ijerph17176218.
4
miR-29b attenuates histone deacetylase-4 mediated podocyte dysfunction and renal fibrosis in diabetic nephropathy.微小RNA-29b减轻组蛋白去乙酰化酶-4介导的糖尿病肾病足细胞功能障碍和肾纤维化。
J Diabetes Metab Disord. 2019 Dec 13;19(1):13-27. doi: 10.1007/s40200-019-00469-0. eCollection 2020 Jun.
5
Targeting mammalian serine/threonine-protein kinase 4 through Yes-associated protein/TEA domain transcription factor-mediated epithelial-mesenchymal transition ameliorates diabetic nephropathy orchestrated renal fibrosis.通过 Yes 相关蛋白/TEA 结构域转录因子介导的上皮间质转化靶向哺乳动物丝氨酸/苏氨酸蛋白激酶 4可改善糖尿病肾病介导的肾纤维化。
Metabolism. 2020 Jul;108:154258. doi: 10.1016/j.metabol.2020.154258. Epub 2020 May 3.
6
Diabetic nephropathy execrates epithelial-to-mesenchymal transition (EMT) via miR-2467-3p/Twist1 pathway.糖尿病肾病通过 miR-2467-3p/Twist1 通路抑制上皮间质转化（EMT）。
Biomed Pharmacother. 2020 May;125:109920. doi: 10.1016/j.biopha.2020.109920. Epub 2020 Feb 9.
7
Visualizing a field of research: A methodology of systematic scientometric reviews.可视化研究领域：系统文献计量学综述方法。
PLoS One. 2019 Oct 31;14(10):e0223994. doi: 10.1371/journal.pone.0223994. eCollection 2019.
8
A small molecule inhibitor MCC950 ameliorates kidney injury in diabetic nephropathy by inhibiting NLRP3 inflammasome activation.小分子抑制剂MCC950通过抑制NLRP3炎性小体激活改善糖尿病肾病中的肾损伤。
Diabetes Metab Syndr Obes. 2019 Aug 2;12:1297-1309. doi: 10.2147/DMSO.S199802. eCollection 2019.
9
New Therapies for the Treatment of Renal Fibrosis.治疗肾纤维化的新疗法。
Adv Exp Med Biol. 2019;1165:625-659. doi: 10.1007/978-981-13-8871-2_31.
10
Myofibroblast in Kidney Fibrosis: Origin, Activation, and Regulation.肌成纤维细胞在肾脏纤维化中的作用：起源、激活和调控。
Adv Exp Med Biol. 2019;1165:253-283. doi: 10.1007/978-981-13-8871-2_12.