从婴儿期到 7 岁的行为抑制发展轨迹：精神病理学的遗传和环境风险的作用。
Developmental trajectories of behavioral inhibition from infancy to age seven: The role of genetic and environmental risk for psychopathology.
机构信息
The Pennsylvania State University, State College, Pennsylvania, USA.
Washington University in St. Louis, St. Louis, Missouri, USA.
出版信息
Child Dev. 2023 Jul-Aug;94(4):e231-e245. doi: 10.1111/cdev.13924. Epub 2023 Apr 5.
The present study leveraged data from a longitudinal adoption study of 361 families recruited between 2003 and 2010 in the United States. We investigated how psychopathology symptoms in birth parents (BP; M = 24.1 years; 50.5-62.9% completed high school) and adoptive parents (AP; M = 37.8 years; 80.9% completed college; 94% mother-father couples) influenced children's behavioral inhibition (BI) trajectories. We used latent growth models of observed BI at 18 and 27 months, and 4.5 and 7 years in a sample of adopted children (Female = 42%, White = 57%, Black = 11%, Multi-racial = 21%, Latinx = 9%). BI generally decreased over time, yet there was substantial variability in these trajectories. Neither BP nor AP psychopathology symptoms independently predicted systematic differences in BI trajectories. Instead, we found that AP internalizing symptoms moderated the effects of BP psychopathology on trajectories of BI, indicating a gene by environment interaction.
本研究利用了一项从 2003 年至 2010 年在美国招募的 361 个家庭的纵向收养研究的数据。我们调查了出生父母（BP；M=24.1 岁；50.5-62.9%完成高中）和养父母（AP；M=37.8 岁；80.9%完成大学；94%为父母双全的家庭）的精神病理学症状如何影响儿童的行为抑制（BI）轨迹。我们使用了观察到的 BI 在 18 个月和 27 个月、4.5 岁和 7 岁的潜变量增长模型，样本包括被收养的儿童（女性=42%，白人=57%，黑人=11%，多种族=21%，拉丁裔=9%）。BI 通常随时间减少，但这些轨迹存在很大的可变性。BP 或 AP 的精神病理学症状都不能独立预测 BI 轨迹的系统差异。相反，我们发现 AP 的内化症状调节了 BP 精神病理学对 BI 轨迹的影响，表明存在基因与环境的相互作用。
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