感染鼠伤寒沙门氏菌的小鼠的粒细胞-巨噬细胞前体细胞和集落刺激因子反应
Granulocyte-macrophage precursor cell and colony-stimulating factor responses of mice infected with Salmonella typhimurium.
作者信息
Trudgett A, McNeill T A, Killen M
出版信息
Infect Immun. 1973 Sep;8(3):450-5. doi: 10.1128/iai.8.3.450-455.1973.
Abstract
The response of granulocyte-macrophage progenitor cells (in vitro colony-forming cells) and of colony-stimulating (CS) factor in serum were studied in mice infected intraperitoneally with 10(3) viable Salmonella typhimurium. Increases in the number of colony-forming cells in marrow and spleen and increases in the serum level of CS factor occurred during the infection. There was no evidence to suggest that progressive infection was associated with failure of macrophage production. Medium rich in CS factor increased the bactericidal activity of macrophages in vitro and it was suggested that CS factor could be involved in macrophage activation.
摘要
对经腹腔感染10³ 活鼠伤寒沙门氏菌的小鼠，研究了粒细胞 - 巨噬细胞祖细胞（体外集落形成细胞）的反应以及血清中集落刺激（CS）因子的情况。感染期间，骨髓和脾脏中集落形成细胞数量增加，血清CS因子水平升高。没有证据表明进行性感染与巨噬细胞生成失败有关。富含CS因子的培养基在体外增加了巨噬细胞的杀菌活性，提示CS因子可能参与巨噬细胞的激活。
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