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系统性红斑狼疮(SLE)患者中抑制性T淋巴细胞功能丧失。

Loss of suppressor T-lymphocyte function in patients with systemic lupus erythematosus (SLE).

作者信息

Morimoto C

出版信息

Clin Exp Immunol. 1978 Apr;32(1):125-33.

Abstract

Immunological reactivity in patients with SLE was studied in vitro trinitrobenzene sulphonate (TNP) specific antibody formation by peripheral blood lymphocytes. Lymphocytes from patients with SLE could produce an increased number of TNP-specific plaque-forming cells (PFC), while no such response could be seen in normal controls. Co-culture of lymphocytes from active SLE patients and normal controls was performed with TNP-Horse red blood cells (TNP-HRBC). The number of PFC by B lymphocytes from active SLE patients was suppressed by T lymphocytes from normal controls. On the other hand, the number of PFC by B lymphocytes from normal controls was increased by T lymphocytes from active SLE patients. Co-culture of lymphocytes from identical twins discordant for SLE was also performed, and the same results were obtained. We further examined the effects of Con A on antibody formation. Con A-treated T lymphocytes from a normal control markedly suppressed TNP-specific PFC by peripheral lymphocytes from active SLE patients. However, Con A-treated T lymphocytes from an active SLE patient did not suppress TNP-specific PFC by lymphocytes from another active SLE patient. These results suggest that active SLE patients showed a loss of suppressor T-lymphocyte function.

摘要

通过外周血淋巴细胞体外三硝基苯磺酸盐(TNP)特异性抗体形成,研究了系统性红斑狼疮(SLE)患者的免疫反应性。SLE患者的淋巴细胞可产生更多数量的TNP特异性空斑形成细胞(PFC),而在正常对照中未观察到这种反应。将活动期SLE患者和正常对照的淋巴细胞与TNP - 马红细胞(TNP - HRBC)进行共培养。正常对照的T淋巴细胞可抑制活动期SLE患者B淋巴细胞产生的PFC数量。另一方面,活动期SLE患者的T淋巴细胞可增加正常对照B淋巴细胞产生的PFC数量。对患SLE的同卵双胞胎的淋巴细胞也进行了共培养,并获得了相同的结果。我们进一步研究了刀豆蛋白A(Con A)对抗体形成的影响。正常对照经Con A处理的T淋巴细胞可显著抑制活动期SLE患者外周淋巴细胞产生的TNP特异性PFC。然而,活动期SLE患者经Con A处理的T淋巴细胞并不能抑制另一名活动期SLE患者淋巴细胞产生的TNP特异性PFC。这些结果表明,活动期SLE患者表现出抑制性T淋巴细胞功能丧失。

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