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小麦激酶和免疫受体形成对炭疽病菌的宿主特异性障碍。

A wheat kinase and immune receptor form host-specificity barriers against the blast fungus.

机构信息

John Innes Centre, Norwich Research Park, Norwich, UK.

Limagrain UK Ltd, Lincolnshire, UK.

出版信息

Nat Plants. 2023 Mar;9(3):385-392. doi: 10.1038/s41477-023-01357-5. Epub 2023 Feb 16.

DOI:10.1038/s41477-023-01357-5
PMID:36797350
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC10027608/
Abstract

Since emerging in Brazil in 1985, wheat blast has spread throughout South America and recently appeared in Bangladesh and Zambia. Here we show that two wheat resistance genes, Rwt3 and Rwt4, acting as host-specificity barriers against non-Triticum blast pathotypes encode a nucleotide-binding leucine-rich repeat immune receptor and a tandem kinase, respectively. Molecular isolation of these genes will enable study of the molecular interaction between pathogen effector and host resistance genes.

摘要

自 1985 年在巴西出现以来,小麦枯萎病已蔓延至整个南美洲,最近又出现在孟加拉国和赞比亚。在这里,我们表明,两个小麦抗性基因 Rwt3 和 Rwt4,作为针对非小麦属枯萎病小种的宿主特异性障碍,分别编码一个核苷酸结合亮氨酸丰富重复免疫受体和一个串联激酶。这些基因的分子分离将使研究病原体效应子和宿主抗性基因之间的分子相互作用成为可能。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/533b/10027608/7eb648a426bd/41477_2023_1357_Fig2_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/533b/10027608/b73c263825e6/41477_2023_1357_Fig1_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/533b/10027608/7eb648a426bd/41477_2023_1357_Fig2_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/533b/10027608/b73c263825e6/41477_2023_1357_Fig1_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/533b/10027608/7eb648a426bd/41477_2023_1357_Fig2_HTML.jpg

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